Accelerated cellular senescence phenotype of GAPDH-depleted human lung carcinoma cells

Biochemical and Biophysical Research Communications

Volumn 411, Issue 2, 2011, Pages 409-415

  Phadke, Manali ^a   Krynetskaia, Natalia ^a   Mishra, Anurag ^a   Krynetskiy, Evgeny ^a  

^a TEMPLE UNIVERSITY SCHOOL OF PHARMACY   (United States)

Author keywords

AMPK; GAPDH; Glycolysis; Lung cancer; Metabolic stress; Senescence

Indexed keywords

BETA GALACTOSIDASE; DELETED IN ESOPHAGEAL CANCER 1 PROTEIN; DNA; GALACTOSIDASE BETA 1; GLYCERALDEHYDE 3 PHOSPHATE DEHYDROGENASE; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; PROTEIN; PROTEIN KINASE LKB1; THREONINE; UNCLASSIFIED DRUG;

ALPHA CHAIN; ARTICLE; CANCER CELL CULTURE; CANCER MORPHOLOGY; CARCINOMA CELL; CELL CYCLE; CELL PROLIFERATION; CONTROLLED STUDY; DNA DAMAGE; ENERGY METABOLISM; ENZYME ACTIVATION; GLYCOLYSIS; HUMAN; HUMAN CELL; LUNG CARCINOMA CELL; NON SMALL CELL LUNG CANCER CELL; PHENOTYPE; PHOSPHORYLATION; PRIORITY JOURNAL; SENESCENCE; STAINING; UPREGULATION;

BETA-GALACTOSIDASE; CARCINOMA, NON-SMALL-CELL LUNG; CELL AGING; CELL LINE, TUMOR; GENE KNOCKDOWN TECHNIQUES; GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE (PHOSPHORYLATING); HUMANS; LUNG NEOPLASMS; TUMOR SUPPRESSOR PROTEINS;

EID: 79960840741     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2011.06.165     Document Type: Article

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