Up-regulation of Kir2.1 by ER stress facilitates cell death of brain capillary endothelial cells

Biochemical and Biophysical Research Communications

Volumn 411, Issue 2, 2011, Pages 293-298

  Kito, Hiroaki ^a   Yamazaki, Daiju ^a,b,c   Ohya, Susumu ^a   Yamamura, Hisao ^a   Asai, Kiyofumi ^c   Imaizumi, Yuji ^a  

^a NAGOYA CITY UNIVERSITY   (Japan)

^b KYOTO UNIVERSITY   (Japan)

^c NAGOYA CITY UNIVERSITY GRADUATE SCHOOL OF MEDICAL SCIENCES   (Japan)

Author keywords

Brain capillary endothelial cell; Cell death; ER stress; Inward rectifier K+ channel; Membrane hyperpolarization; Tunicamycin

Indexed keywords

CALCIUM ION; CATION CHANNEL; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR2.1; TUNICAMYCIN;

ANIMAL CELL; ARTICLE; BRAIN CAPILLARY; BRAIN CAPILLARY ENDOTHELIAL CELL; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CELL DEATH; CELL STRESS; CONTROLLED STUDY; ENDOPLASMIC RETICULUM STRESS; ENDOTHELIUM CELL; GLYCOSYLATION; HYPERPOLARIZATION; MEMBRANE STEADY POTENTIAL; NONHUMAN; PRIORITY JOURNAL; UPREGULATION;

ANIMALS; BRAIN; CAPILLARIES; CATTLE; CELL LINE; ENDOPLASMIC RETICULUM; ENDOTHELIUM, VASCULAR; POTASSIUM CHANNELS, INWARDLY RECTIFYING; STRESS, PHYSIOLOGICAL; TUNICAMYCIN; UP-REGULATION;

BOVINAE;

EID: 79960840653     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2011.06.128     Document Type: Article

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