Mouse apoA-II amyloid fibrils deposit in skeletal muscle and exhibit amyloidosisenhancing activity

Amyloid

Volumn 18, Issue SUPPL. 1, 2011, Pages 42-44

  Higuchi, K ^a   Qian, J ^a   Yan, J ^a   Ge, F ^a   Zhang, B ^a   Fu, X ^a   Tomozawa, H ^a   Sawashita, J ^a   Mori, M ^a  

^a SHINSHU UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID; APOLIPOPROTEIN A2; CONGO RED;

ALLELE; AMYLOIDOSIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BACK MUSCLE; BLOOD VESSEL; BREAST SURGERY; CONNECTIVE TISSUE; CONTROLLED STUDY; ELECTRON MICROSCOPY; ENDOMYSIUM; FEMALE; FORELIMB; HEART; HINDLIMB; IMMUNOHISTOCHEMISTRY; INTERSTITIUM; INTESTINE; LIVER; LONGISSIMUS MUSCLE; LUNG; MOUSE; MUSCLE BLOOD VESSEL; NONHUMAN; PATHOGENESIS; PECTORAL MUSCLE; PELVIC GIRDLE; PRIORITY JOURNAL; PROTEIN ISOLATION; PROTEIN LOCALIZATION; PROTEIN STRUCTURE; QUADRICEPS FEMORIS MUSCLE; SENILE AMYLOIDOSIS; SKELETAL MUSCLE; SKIN; SPLEEN; STOMACH; TISSUE DISTRIBUTION; TONGUE; TRANSGENIC MOUSE; TRICEPS BRACHII MUSCLE; WESTERN BLOTTING;

EID: 79960023257     PISSN: 13506129     EISSN: 17442818     Source Type: Journal    
DOI: 10.3109/13506129.2011.574354     Document Type: Article

References (10)

1. Qian J, Yan J, Ge F, Zhang B, Fu X, Tomozawa H, Sawashita J, Mori M, Higuchi K. Mouse senile amyloid fibrils deposited in skeletal muscle exhibit amyloidosis-enhancing activity. PLoS Pathog 2010;6:e1000914.
2. Xing Y, Nakamura A, Korenaga T, Guo Z, Yao J, Fu X, Matsushita T, Kogishi K, Hosokawa M, Kametani F, et al. Induction of protein conformational change in mouse senile amyloidosis. J Biol Chem 2002;277:33164-33169. (Pubitemid 34984834)
3. Zhang H, Sawashita J, Fu X, Korenaga T, Yan J, Mori M, Higuchi K. Transmissibility of mouse AApoAII amyloid fibrils: inactivation by physical and chemical methods. FASEB J 2006;20:1012-1014.
4. Ge F, Yao J, Fu X, Guo Z, Yan J, Zhang B, Zhang H, Tomozawa H, Miyazaki J, Sawashita J, et al. Amyloidosis in transgenic mice expressing murine amyloidogenic apolipoprotein A-II (Apoa2c). Lab Invest 2007;87:633-643. (Pubitemid 46920103)
5. Takeshita S, Hosokawa M, Irino M, Higuchi K, Shimizu K, Yasuhira K, Takeda T. Spontaneous age-associated amyloidosis in senescence-accelerated mouse (SAM). Mech Ageing Dev 1982;20:13-23. (Pubitemid 13255124)
6. Bosque PJ, Ryou C, Telling G, Peretz D, Legname G, DeArmond SJ, Prusiner SB. Prions in skeletal muscle. Proc Natl Acad Sci USA 2002;99:3812-3817. (Pubitemid 34252188)
7. Andréoletti O, Simon S, Lacroux C, Morel N, Tabouret G, Chabert A, Lugan S, Corbière F, Ferré P, Foucras G, et al. PrPSc accumulation in myocytes from sheep incubating natural scrapie. Nat Med 2004;10:591-593. (Pubitemid 38796849)
8. Angers RC, Browning SR, Seward TS, Sigurdson CJ, Miller MW, Hoover EA, Telling GC. Prions in skeletal muscles of deer with chronic wasting disease. Science 2006;311:1117.
9. Zhang B, Une Y, Fu X, Yan J, Ge F, Yao J, Sawashita J, Mori M, Tomozawa H, Kametani F, et al. Fecal transmission of AA amyloidosis in the cheetah contributes to high incidence of disease. Proc Natl Acad Sci USA 2008;105:7263-7268. (Pubitemid 351754466)
10. Yoshida T, Zhang P, Fu X, Higuchi K, Ikeda S. Slaughtered aged cattle might be one dietary source exhibiting amyloid enhancing factor activity. Amyloid 2009;16:25-31.