Loss of glial fibrillary acidic protein marginally accelerates disease progression in a SOD1 H46R transgenic mouse model of ALS

Neuroscience Research

Volumn 70, Issue 3, 2011, Pages 321-329

  Yoshii, Yasuhiro ^a   Otomo, Asako ^b   Pan, Lei ^b   Ohtsuka, Masato ^b,d   Hadano, Shinji ^b,c,d  

^a TOHO UNIVERSITY OMORI MEDICAL CENTER   (Japan)

^b TOKAI UNIVERSITY SCHOOL OF MEDICINE   (Japan)

^c TOKAI UNIVERSITY   (Japan)

^d TOKAI UNIVERSITY   (Japan)

Author keywords

Allograft inflammatory factor 1; Amyotrophic lateral sclerosis; Astroglia; Cu Zn superoxide dismutase; Glial fibrillary acidic protein; Microglia; Vimentin

Indexed keywords

ALLOGRAFT INFLAMMATORY FACTOR 1; COPPER ZINC SUPEROXIDE DISMUTASE; GLIAL FIBRILLARY ACIDIC PROTEIN; VIMENTIN;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; ASTROCYTE; ASTROCYTOSIS; CELL ACTIVATION; CONTROLLED STUDY; DISEASE COURSE; GLIA CELL; LIFESPAN; MOTOR NEURON DISEASE; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; SPINAL CORD; TRANSGENIC MOUSE;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; ASTROCYTES; DISEASE MODELS, ANIMAL; DISEASE PROGRESSION; FEMALE; GLIAL FIBRILLARY ACIDIC PROTEIN; HUMANS; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, TRANSGENIC; NEUROGLIA; SPINAL CORD; SUPEROXIDE DISMUTASE;

EID: 79957598029     PISSN: 01680102     EISSN: 18728111     Source Type: Journal    
DOI: 10.1016/j.neures.2011.03.006     Document Type: Article

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