Distinct different contributions of the alternative and classical complement activation pathway for the innate host response during sepsis

Journal of Immunology

Volumn 186, Issue 5, 2011, Pages 3066-3075

  Dahlke, Katja ^a   Wrann, Christiane D ^b   Sommerfeld, Oliver ^a   Soßdorf, Maik ^a   Recknagel, Peter ^a   Sachse, Svea ^a   Winter, Sebastian W ^b   Klos, Andreas ^b   Stahl, Gregory L ^c   Ma, Yuanyuan Xu ^d   Claus, Ralf A ^a   Reinhart, Konrad ^a   Bauer, Michael ^a   Riedemann, Niels C ^a  

^a JENA UNIVERSITY HOSPITAL   (Germany)

^b HANNOVER MEDICAL SCHOOL   (Germany)

^c BRIGHAM AND WOMEN S HOSPITAL   (United States)

^d The University of Alabama at Birmingham   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALTERNATIVE COMPLEMENT PATHWAY C3 C5 CONVERTASE; COMPLEMENT COMPONENT C1Q; GAMMA INTERFERON; INTERLEUKIN 6; MONOCYTE CHEMOTACTIC PROTEIN; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BACTERIAL CLEARANCE; BACTERIAL GROWTH; CECAL LIGATION; CECAL PUNCTURE; CECUM; CONTROLLED STUDY; INFLAMMATION; INNATE IMMUNITY; KNOCKOUT MOUSE; MOUSE; NEUTROPHIL; NONHUMAN; ONSET AGE; ORGAN INJURY; PRIORITY JOURNAL; PYLORUS LIGATION; SEPSIS; SURVIVAL;

ANIMALS; BACTERIAL LOAD; CECUM; COMPLEMENT ACTIVATION; COMPLEMENT C1Q; COMPLEMENT FACTOR D; COMPLEMENT PATHWAY, ALTERNATIVE; COMPLEMENT PATHWAY, CLASSICAL; IMMUNITY, INNATE; KIDNEY; LIGATION; LIVER; LUNG; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PUNCTURES; SHOCK, SEPTIC; SURVIVAL ANALYSIS;

EID: 79952743025     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1002741     Document Type: Article

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