-
1
-
-
0035282458
-
Selective recognition of methylated lysine 9 on histone H3 by the HP1 chromo domain
-
DOI 10.1038/35065138
-
Bannister, A. J., Zegerman, P., Partridge, J. F., Miska, E. A., Thomas, J. O., Allshire, R. C., and Kouzarides, T. (2001). Selective recognition of methylated lysine 9 on histone H3 by the HP1 (Pubitemid 32225848)
-
(2001)
Nature
, vol.410
, Issue.6824
, pp. 120-124
-
-
Bannister, A.J.1
Zegerman, P.2
Partridge, J.F.3
Miska, E.A.4
Thomas, J.O.5
Allshire, R.C.6
Kouzarides, T.7
-
2
-
-
70349975711
-
JAK2 phosphorylates histone H3Y41 and excludes HP1alpha from chromatin
-
Dawson, M. A., Bannister, A. J., Göttgens, B., Foster, S. D., Bartke, T., Green, A. R., and Kouzarides, T. (2009). JAK2 phosphorylates histone H3Y41 and excludes HP1alpha from chromatin. Nature 461, 819-822.
-
(2009)
Nature
, vol.461
, pp. 819-822
-
-
Dawson, M.A.1
Bannister, A.J.2
Göttgens, B.3
Foster, S.D.4
Bartke, T.5
Green, A.R.6
Kouzarides, T.7
-
3
-
-
0037183856
-
Establishment and maintenance of a heterochromatin domain
-
Hall, I. M., Shankaranarayana, G. D., Noma, K., Ayoub, N., Cohen, A., and Grewal, S. I. S. (2002). Establishment and maintenance of a heterochromatin domain. Science 297, 2232-2237.
-
(2002)
Science
, vol.297
, pp. 2232-2237
-
-
Hall, I.M.1
Shankaranarayana, G.D.2
Noma, K.3
Ayoub, N.4
Cohen, A.5
Grewal, S.I.S.6
-
4
-
-
33846216449
-
Jak2: normal function and role in hematopoietic disorders
-
DOI 10.1016/j.gde.2006.12.009, PII S0959437X06002437, Genetic and Cellular mechanisms of oncogenesis
-
Ihle, J. N., and Gilliland, D. G. (2007). JAK2: normal function and role in hematopoietic disorders. Curr. Opin. Genet. Dev. 17, 8-14. (Pubitemid 46109298)
-
(2007)
Current Opinion in Genetics and Development
, vol.17
, Issue.1
, pp. 8-14
-
-
Ihle, J.N.1
Gilliland, D.G.2
-
5
-
-
1342289322
-
Activation of the T-Cell Oncogene LMO2 after Gene Therapy for X-Linked Severe Combined Immunodeficiency
-
DOI 10.1056/NEJMra032207
-
McCormack, M. P., and Rabbitts, T. H. (2004). Activation of the T-cell oncogene LMO2 after gene therapy for X-linked severe combined immunodeficiency. N. Engl. J. Med. 350, 913-922. (Pubitemid 38252539)
-
(2004)
New England Journal of Medicine
, vol.350
, Issue.9
, pp. 913-922
-
-
McCormack, M.P.1
Rabbitts, T.H.2
-
6
-
-
18244432009
-
Jak2 deficiency defines an essential developmental checkpoint in definitive hematopoiesis
-
DOI 10.1016/S0092-8674(00)81168-X
-
Neubauer, H., Cumano, A., Muller, M., Wu, H., Huffstadt, U., and Pfeffer, K. (1998). Jak2 deficiency defines an essential developmental checkpoint in definitive hematopoiesis. Cell 93, 397-409. (Pubitemid 28232084)
-
(1998)
Cell
, vol.93
, Issue.3
, pp. 397-409
-
-
Neubauer, H.1
Cumano, A.2
Muller, M.3
Wu, H.4
Huffstadt, U.5
Pfeffer, K.6
-
7
-
-
0032076542
-
Jak2 is essential for signaling through a variety of cytokine receptors
-
DOI 10.1016/S0092-8674(00)81167-8
-
Parganas, E., Wang, D., Stravopodis, D., Topham, D. J., Marine, J.-C., Teglund, S., Vanin, E. F., Bodner, S., Colamonici, O. R., van Deursen, J. M., et al. (1998). Jak2 is essential for signaling through a variety of cytokine receptors. Cell 93, 385-395. (Pubitemid 28232083)
-
(1998)
Cell
, vol.93
, Issue.3
, pp. 385-395
-
-
Parganas, E.1
Wang, D.2
Stravopodis, D.3
Topham, D.J.4
Marine, J.-C.5
Teglund, S.6
Vanin, E.F.7
Bodner, S.8
Colamonici, O.R.9
Van Deursen, J.M.10
Grosveld, G.11
Ihle, J.N.12
-
8
-
-
51649087754
-
JAK2 stimulates homologous recombination and genetic instability: Potential implication in the heterogeneity of myeloproliferative disorders
-
Plo, I., Nakatake, M., Malivert, L., de Villartay, J. P., Giraudier, S., Villeval, J. L., Wiesmuller, L., and Vainchenker, W. (2008). JAK2 stimulates homologous recombination and genetic instability: potential implication in the heterogeneity of myeloproliferative disorders. Blood 112, 1402-1412.
-
(2008)
Blood
, vol.112
, pp. 1402-1412
-
-
Plo, I.1
Nakatake, M.2
Malivert, L.3
De Villartay, J.P.4
Giraudier, S.5
Villeval, J.L.6
Wiesmuller, L.7
Vainchenker, W.8
-
9
-
-
0033639119
-
Stat5 is essential for the myelo-and lymphoproliferative disease induced by TEL/JAK2
-
Schwaller, J., Parganas, E., Wang, D., Cain, D., Aster, J. C., Williams, I. R., Lee, C. K., Gerthner, R., Kitamura, T., Frantsve, J., et al. (2000). Stat5 is essential for the myelo-and lymphoproliferative disease induced by TEL/JAK2. Mol. Cell 6, 693-704.
-
(2000)
Mol. Cell.
, vol.6
, pp. 693-704
-
-
Schwaller, J.1
Parganas, E.2
Wang, D.3
Cain, D.4
Aster, J.C.5
Williams, I.R.6
Lee, C.K.7
Gerthner, R.8
Kitamura, T.9
Frantsve, J.10
-
10
-
-
33748308794
-
JAK signaling globally counteracts heterochromatic gene silencing
-
DOI 10.1038/ng1860, PII NG1860
-
Shi, S., Calhoun, H. C., Xia, F., Li, J., Le, L., and Li, W. X. (2006). JAK signaling globally counteracts heterochromatin gene silencing. Nat. Genet. 38, 1071-1076. (Pubitemid 44325935)
-
(2006)
Nature Genetics
, vol.38
, Issue.9
, pp. 1071-1076
-
-
Shi, S.1
Calhoun, H.C.2
Xia, F.3
Li, J.4
Le, L.5
Li, W.X.6
-
11
-
-
25444516768
-
JAK2 mutations in myeloproliferative disorders
-
Tefferi, A., Lasho, T. L., and Gilliland, G. (2005). JAK2 mutations in myeloproliferative disorders. N. Engl. J. Med. 353, 1416-1417.
-
(2005)
N. Engl. J. Med.
, vol.353
, pp. 1416-1417
-
-
Tefferi, A.1
Lasho, T.L.2
Gilliland, G.3
|