RMI1 attenuates tumor development and is essential for early embryonic survival

Molecular Carcinogenesis

Volumn 50, Issue 2, 2011, Pages 80-88

  Chen, H ^a   You, M J ^a   Jiang, Y ^a   Wang, W ^b   Li, L ^a  

^a UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

^b NATIONAL INSTITUTE ON AGING   (United States)

Author keywords

Bloom syndrome; Genomic instability; Radiation; RMI1; Tumor development

Indexed keywords

BLOOM SYNDROME HELICASE; PROTEIN P53; PROTEIN P75; RECQ HELICASE;

ANIMAL EXPERIMENT; ARTICLE; BLOOM SYNDROME; CANCER SURVIVAL; CARCINOGENESIS; COMPARATIVE STUDY; CONTROLLED STUDY; EMBRYO DEVELOPMENT; GENOMIC INSTABILITY; HETEROZYGOTE; IONIZING RADIATION; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL;

ANIMALS; BLASTOCYST; CARCINOMA; CARRIER PROTEINS; CELL LINE, TUMOR; CELL TRANSFORMATION, NEOPLASTIC; EMBRYO LOSS; EMBRYONIC DEVELOPMENT; FEMALE; GAMMA RAYS; GENES, LETHAL; HUMANS; LYMPHOMA; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEOPLASMS, RADIATION-INDUCED; NUCLEAR PROTEINS; OSTEOSARCOMA; RECQ HELICASES;

MUS;

EID: 78651439805     PISSN: 08991987     EISSN: 10982744     Source Type: Journal    
DOI: 10.1002/mc.20694     Document Type: Article

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