Fusion genes in cord blood

Blood

Volumn 117, Issue 1, 2011, Pages 369-370

  Greaves, Mel ^a   Colman, Susan M ^a   Kearney, Lyndal ^a   Ford, Anthony M ^a  

^a INSTITUTE OF CANCER RESEARCH   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

CD19 ANTIBODY; COMPLEMENTARY DNA; MESSENGER RNA; TRANSCRIPTION FACTOR ETV6; TRANSCRIPTION FACTOR RUNX1;

ACUTE LYMPHOBLASTIC LEUKEMIA; CLONE; DNA MICROARRAY; FETUS; FETUS DEVELOPMENT; FLUORESCENCE IN SITU HYBRIDIZATION; FUSION GENE; HUMAN; LETTER; PRIORITY JOURNAL; PROTEIN EXPRESSION; RESCREENING; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; RNA EXTRACTION; UMBILICAL CORD BLOOD;

EID: 78650982149     PISSN: 00064971     EISSN: 15280020     Source Type: Journal    
DOI: 10.1182/blood-2010-09-309351     Document Type: Letter

References (4)

1. Lausten-Thomsen U, Madsen HO, Vestergaard TR, Hjalgrim H, Nersting J, Schmiegelow K. Prevalence of t(12;21)[ETV6-RUNX1]-positive cells in healthy neonates. Blood. 2011;117(1):186-189.
2. Mori H, Colman SM, Xiao Z, et al. Chromosome translocations and covert leukemic clones are generated during normal fetal development. Proc Natl Acad Sci U S A. 2002;99(12):8242-8247.
3. Greaves MF, Wiemels J. Origins of chromosome translocations in childhood leukaemia. Nat Rev Cancer. 2003;3(9):639-649. (Pubitemid 37328819)
4. Zuna J, Madzo J, Krejci O, et al. ETV6/RUNX1 (TEL/AML1) is a frequent prenatal first hit in childhood leukemia [letter]. Blood. 2011;117(1):368-369.