Deficiency of methionine sulfoxide reductase A causes cellular dysfunction and mitochondrial damage in cardiac myocytes under physical and oxidative stresses

Biochemical and Biophysical Research Communications

Volumn 402, Issue 4, 2010, Pages 608-613

  Nan, Changlong ^a   Li, Yuejin ^a   Jean Charles, Pierre Yves ^a   Chen, Guozhen ^a   Kreymerman, Alexander ^b   Prentice, Howard ^a   Weissbach, Herbert ^b   Huang, Xupei ^a,b  

^a FLORIDA ATLANTIC UNIVERSITY   (United States)

^b FLORIDA ATLANTIC UNIVERSITY   (United States)

Author keywords

Cardiac contractility; Knockout mice; Methionine sulfoxide reductase; Mitochondria; Protein oxidation

Indexed keywords

CALCIUM ION; HYDROGEN PEROXIDE; METHIONINE SULFOXIDE REDUCTASE A;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM TRANSPORT; CELL STIMULATION; CELLULAR STRESS RESPONSE; CONTROLLED STUDY; ELECTRON MICROSCOPY; FEMALE; HEART DISEASE; HEART FUNCTION; HEART MITOCHONDRION; HEART MUSCLE CELL; MALE; MOUSE; MUSCLE FIBER CONTRACTION; NONHUMAN; OXIDATIVE STRESS; PHYSICAL STRESS; PRIORITY JOURNAL; PROTEIN FUNCTION; WILD TYPE;

ANIMALS; CALCIUM; METHIONINE SULFOXIDE REDUCTASES; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOCHONDRIA, HEART; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; OXIDATIVE STRESS; STRESS, MECHANICAL;

MUS;

EID: 78649444069     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2010.10.064     Document Type: Article

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