Sequential alterations in Akt, GSK3β, and calcineurin signalling in the mouse left ventricle after thoracic aortic constriction

Canadian Journal of Physiology and Pharmacology

Volumn 88, Issue 11, 2010, Pages 1093-1101

  Prévilon, Miresta ^a   Pezet, Mylene ^b   Dachez, Céline ^a   Mercadier, Jean Jacques ^a,b,c   Rouet Benzineb, Patricia ^a  

^a UNIVERSITÉ PARIS DESCARTES   (France)

^b BICHAT HOSPITAL   (France)

^c ASSISTANCE PUBLIQUE HÔPITAUX DE PARIS   (France)

Author keywords

Akt; Calcineurin; Calcium handling proteins; Cardiac hypertrophy; Cell signalling; Glycogen synthase kinase 3 ; Heart failure; Pressure overload

Indexed keywords

CALCINEURIN; CALCIUM ION; GLYCOGEN SYNTHASE KINASE 3BETA; PROTEIN KINASE; PROTEIN KINASE B;

ANIMAL MODEL; AORTA CONSTRICTION; ARTICLE; CONTROLLED STUDY; DISEASE COURSE; ENZYME ACTIVATION; ENZYME INACTIVATION; GENE EXPRESSION; HEART FAILURE; HEART LEFT VENTRICLE HYPERTROPHY; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; RECEPTOR DOWN REGULATION; RECEPTOR UPREGULATION; SIGNAL TRANSDUCTION; TRANSVERSE AORTA CONSTRICTION;

ANIMALS; AORTA, THORACIC; CALCINEURIN; CALCIUM; GLYCOGEN SYNTHASE KINASE 3; HEART FAILURE; HYPERTROPHY, LEFT VENTRICULAR; MALE; MICE; NFATC TRANSCRIPTION FACTORS; PROTO-ONCOGENE PROTEINS C-AKT; SIGNAL TRANSDUCTION;

AKT; CALCINEURIN; CARDIAC HYPERTROPHY; CELL SIGNALLING; GLYCOGEN SYNTHASE KINASE-3; HEART FAILURE; PRESSURE OVERLOAD;

ACTIVATION ANALYSIS; BIOMECHANICS; BLOOD VESSELS; CALCIUM; HEART; PATHOLOGY; PROTEINS;

ENZYME ACTIVITY;

EID: 78549243374     PISSN: 00084212     EISSN: 12057541     Source Type: Journal    
DOI: 10.1139/Y10-087     Document Type: Article

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