Myocardial stress remodelling after regional infarction is independent of glycogen synthase kinase-3 inactivation

Journal of Molecular and Cellular Cardiology

Volumn 49, Issue 5, 2010, Pages 897-900

  Webb, Ian G ^a   Sicard, Pierre ^a   Clark, James E ^a   Redwood, Simon ^a   Marber, Michael S ^a  

^a KING'S COLLEGE LONDON   (United Kingdom)

Author keywords

GSK 3; Myocardial infarction; Remodelling

Indexed keywords

ALANINE; GLYCOGEN SYNTHASE KINASE 3; GLYCOGEN SYNTHASE KINASE 3ALPHA; GLYCOGEN SYNTHASE KINASE 3BETA;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; GENOTYPE; HEART FUNCTION; HEART LEFT VENTRICLE CONTRACTILITY; HEART LEFT VENTRICLE VOLUME; HEART LEFT VENTRICLE WALL; HEART STRESS; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE REMODELING; HEART WEIGHT; KNOCKOUT MOUSE; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANIMALS; ENZYME ACTIVATION; GLYCOGEN SYNTHASE KINASE 3; MICE; MYOCARDIAL INFARCTION; MYOCARDIUM; PHOSPHORYLATION; STRESS, PHYSIOLOGICAL; SURVIVAL ANALYSIS; VENTRICULAR REMODELING;

ANIMALIA; MUS;

EID: 77957224804     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2010.07.021     Document Type: Article

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