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Volumn 12, Issue 4, 2010, Pages

Could the expression of CD86 and FcγRIIB on B cells be functionally related and involved in driving rheumatoid arthritis?

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; CD86 ANTIGEN; FC RECEPTOR IIA; FC RECEPTOR IIB; BIOLOGICAL MARKER; CD86 PROTEIN, HUMAN; FC RECEPTOR; FCGR2B PROTEIN, HUMAN;

EID: 77955497834     PISSN: 14786354     EISSN: 14786362     Source Type: Journal    
DOI: 10.1186/ar3092     Document Type: Editorial
Times cited : (6)

References (11)
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    • B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and FcγRIIb, which are modulated by anti-tumor necrosis factor therapy
    • 2888223, 20398308, 10.1186/ar2985, for Millenium Nucleus on Immunology and Immunotherapy P-07-088-F
    • Catalán D, Aravena O, Sabugo F, Wurmann P, Soto L, Kalergis AM, Cuchacovich M, Aguillon JC, . for Millenium Nucleus on Immunology and Immunotherapy P-07-088-F B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and FcγRIIb, which are modulated by anti-tumor necrosis factor therapy. Arthritis Res Ther 2010, 12:R68. 2888223, 20398308, 10.1186/ar2985, for Millenium Nucleus on Immunology and Immunotherapy P-07-088-F.
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  • 2
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  • 3
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  • 6
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    • Petkova SB, Konstantinov KN, Sproule TJ, Lyons BL, Awwami MA, Roopenian DC. Human antibodies induce arthritis in mice deficient in the low-affinity inhibitory IgG receptor FcÎ3RIIB. J Exp Med 2006, 203:275-280. 2118221, 16476768, 10.1084/jem.20051951.
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  • 9
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.