Axon initial segment dysfunction in a mouse model of genetic epilepsy with febrile seizures plus

Journal of Clinical Investigation

Volumn 120, Issue 8, 2010, Pages 2661-2671

  Wimmer, Verena C ^a   Reid, Christopher A ^a   Mitchell, Suzanne ^a   Richards, Kay L ^a   Scaf, Byron B ^a   Leaw, Bryan T ^a   Hill, Elisa L ^a   Royeck, Michel ^b   Horstmann, Marie Therese ^b,c   Cromer, Brett A ^a   Davies, Philip J ^a   Xu, Ruwei ^a   Lerche, Holger ^d,e   Berkovic, Samuel F ^a   Beck, Heinz ^b   Petrou, Steven ^a  

^a UNIVERSITY OF MELBOURNE   (Australia)

^b UNIVERSITY HOSPITAL BONN   (Germany)

^c UNIVERSITY OF BONN   (Germany)

^d UNIVERSITY OF ULM   (Germany)

^e UNIVERSITY OF TÜBINGEN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

SODIUM CHANNEL; SODIUM CHANNEL BETA1; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; CORE TEMPERATURE; DISEASE MODEL; EPILEPSY; EPILEPTIC DISCHARGE; FEBRILE CONVULSION; GENETIC EPILEPSY WITH FEBRILE SEIZURES PLUS; HEAT SENSITIVITY; MOUSE; NERVE CELL EXCITABILITY; NERVE FIBER MEMBRANE; NONHUMAN; PRIORITY JOURNAL; PYRAMIDAL NERVE CELL; SEIZURE SUSCEPTIBILITY; SYNDROME;

ANIMALS; AXONS; DISEASE MODELS, ANIMAL; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MUTATION; PHENOTYPE; PROTEIN SUBUNITS; SEIZURES, FEBRILE; SODIUM CHANNELS;

EID: 77955294896     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI42219     Document Type: Article

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