Hypoxia-induced alteration of mitochondrial genes in cardiomyocytes: Role of Bnip3 and Pdk1

Shock

Volumn 34, Issue 2, 2010, Pages 169-175

  Jian, Bixi ^a   Wang, Deli ^b   Chen, Dongquan ^c   Voss, Joachim ^d   Chaudry, Irshad ^a,e   Raju, Raghavan ^a,e  

^a Kentucky   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^c WEST VIRGINIA UNIVERSITY   (United States)

^d UNIVERSITY OF WASHINGTON   (United States)

^e University of Alabama at Birmingham   (United States)

Author keywords

apoptosis; custom chip; Heart; microarray; mitochip

Indexed keywords

FATTY ACID; FRUCTOSE BISPHOSPHATE ALDOLASE; FRUCTOSE BISPHOSPHATE ALDOLASE 3; GLUCOSE; HYPOXIA INDUCIBLE FACTOR 1ALPHA; MESSENGER RNA; MITOCHONDRIAL DNA; MITOCHONDRIAL PROTEIN; NUCLEAR PROTEIN; PHOSPHOINOSITIDE DEPENDENT PROTEIN KINASE 1; PROTEIN BNIP3; TRANSFER RNA; UNCLASSIFIED DRUG; BNIP3 PROTEIN, MOUSE; MEMBRANE PROTEIN; PROTEIN SERINE THREONINE KINASE; PYRUVATE DEHYDROGENASE (ACETYL-TRANSFERRING) KINASE;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL FUNCTION; CELL HYPOXIA; CELL ISOLATION; CELL STRUCTURE; CONTROLLED STUDY; FATTY ACID METABOLISM; GENE; GENE EXPRESSION; GENE EXPRESSION PROFILING; GENETIC TRANSCRIPTION; GLUCOSE METABOLISM; HEART MITOCHONDRION; HEART MUSCLE CELL; MICROCHIP ANALYSIS; MOLECULAR PROBE; NONHUMAN; NUCLEOTIDE SEQUENCE; OXIDATIVE STRESS; PROTEIN PHOSPHORYLATION; RECEPTOR UPREGULATION; RODENT MITOCHONDRIAL GENE CHIP; SIGNAL TRANSDUCTION; ANIMAL; ANOXIA; C57BL MOUSE; DRUG EFFECTS; GLYCOLYSIS; METABOLISM; MOUSE; PATHOPHYSIOLOGY; PHYSIOLOGY;

ANIMALS; ANOXIA; DNA, MITOCHONDRIAL; GENE EXPRESSION PROFILING; GLYCOLYSIS; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MITOCHONDRIAL PROTEINS; MYOCYTES, CARDIAC; PROTEIN-SERINE-THREONINE KINASES; RNA, MESSENGER;

EID: 77954966737     PISSN: 10732322     EISSN: None     Source Type: Journal    
DOI: 10.1097/SHK.0b013e3181cffe7d     Document Type: Article

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