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Volumn 429, Issue 2, 2010, Pages 335-345

Attenuation of the hypoxia-induced protein kinase Cδ interaction with the 'd' subunit of F1Fo-ATP synthase in neonatal cardiac myocytes: Implications for energy preservation and survival

Author keywords

Cardiac injury; F1Fo ATP synthase; Hypoxia; Mitochondrion; Oxidative phosphorylation; Protein kinase C (PKC)

Indexed keywords

ATP LEVEL; ATP SYNTHASE; ATP-ASE ACTIVITY; CARDIAC INJURY; CARDIAC MYOCYTES; CO-IMMUNOPRECIPITATIONS; EXTRACELLULAR CONCENTRATION; HIV-TAT; HYPOXIC CONDITION; INDUCED LOSS; INDUCED PROTEINS; INHIBITORY EFFECT; MITOCHONDRIAL MEMBRANE POTENTIAL; OXIDATIVE PHOSPHORYLATION; OXYGEN DEPRIVATION; PRE-INCUBATION; PROTEIN KINASE C; PROTEIN TRANSDUCTION; REPERFUSION; SYNTHASES; TRANSACTIVATORS;

EID: 77954733228     PISSN: 02646021     EISSN: 14708728     Source Type: Journal    
DOI: 10.1042/BJ20091927     Document Type: Article
Times cited : (9)

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