Doxorubicin coupled to penetratin promotes apoptosis in CHO cells by a mechanism involving c-Jun NH2-terminal kinase

Biochemical and Biophysical Research Communications

Volumn 396, Issue 4, 2010, Pages 908-914

  Aroui, Sonia ^a   Mili, Donia ^a   Brahim, Souhir ^a   Waard, Michel De ^b   Kenani, Abderraouf ^a  

^a UNIVERSITY OF MONASTIR   (Tunisia)

^b GRENOBLE INSTITUT DES NEUROSCIENCES   (France)

Author keywords

Apoptosis; Doxorubicin; JNK; Mitochondrial apoptotic pathway; Penetratin

Indexed keywords

CASPASE 3; CASPASE 7; CELL PENETRATING PEPTIDE; CYTOCHROME C; DOXORUBICIN; PROTEIN BAX; REACTIVE OXYGEN METABOLITE; STRESS ACTIVATED PROTEIN KINASE; TUMOR NECROSIS FACTOR RELATED APOPTOSIS INDUCING LIGAND;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; CELL VIABILITY; CHO CELL; CHROMATIN CONDENSATION; CONCENTRATION RESPONSE; CONTROLLED STUDY; CYTOTOXICITY TEST; EMBRYO; ENZYME ACTIVATION; IC 50; MITOCHONDRION; NONHUMAN; OLIGOMERIZATION; PHENOTYPE; PRIORITY JOURNAL; RAT;

ANIMALS; ANTIBIOTICS, ANTINEOPLASTIC; APOPTOSIS; CARRIER PROTEINS; CHO CELLS; CRICETINAE; CRICETULUS; DOXORUBICIN; JNK MITOGEN-ACTIVATED PROTEIN KINASES; REACTIVE OXYGEN SPECIES; RECEPTORS, TNF-RELATED APOPTOSIS-INDUCING LIGAND;

EID: 77953475811     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2010.05.020     Document Type: Article

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