Tumor necrosis factor-α produced in cardiomyocytes mediates a predominant myocardial inflammatory response to stretch in early volume overload

Journal of Molecular and Cellular Cardiology

Volumn 49, Issue 1, 2010, Pages 70-78

  Chen, Yuanwen ^a,b   Pat, Betty ^b   Zheng, Junying ^b   Cain, Laura ^b   Powell, Pamela ^b   Shi, Ke ^b   Sabri, Abdelkarim ^d   Husain, Ahsan ^b,e   Dell'Italia, Louis J ^b,c  

^a SHANGHAI JIAO TONG UNIVERSITY SCHOOL OF MEDICINE   (China)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^c Department of Veteran Affairs ^*  (United States)

^d TEMPLE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^e EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Cardiac myocytes; Mast cells; Myocardial inflammation; Tumor necrosis factor ; Volume overload

Indexed keywords

CD45 ANTIGEN; COLLAGEN; GELATINASE A; INFLIXIMAB; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTOCAVAL FISTULA; APOPTOSIS; ARTICLE; CARDITIS; CELL INFILTRATION; COLLAGEN DEGRADATION; CONTROLLED STUDY; CYTOKINE PRODUCTION; ENZYME ACTIVATION; GENE EXPRESSION PROFILING; GENE REGULATORY NETWORK; HEART DISEASE; HEART LEFT VENTRICLE; HEART LEFT VENTRICLE OVERLOAD; HEART MUSCLE CELL; HEART VENTRICLE FUNCTION; INFLAMMATION; MAST CELL; MUSCLE STRETCHING; NEUTROPHIL; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; RAT; UPREGULATION; WESTERN BLOTTING;

ANIMALS; APOPTOSIS; HEART; MAST CELLS; MATRIX METALLOPROTEINASE 2; MATRIX METALLOPROTEINASES; MYOCARDITIS; MYOCARDIUM; MYOCYTES, CARDIAC; RATS; TUMOR NECROSIS FACTOR-ALPHA; UP-REGULATION; VENTRICULAR REMODELING;

FISTULA; RATTUS;

EID: 77953358639     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2009.12.013     Document Type: Article

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