SCOPUS 정보 검색 플랫폼

Volumn 362, Issue 14, 2010, Pages 1348-1350

Large islets, beta-cell proliferation, and a glucokinase mutation

(12) Kassem, Sameer a Heyman, Maayan a Glaser, Benjamin a Bhandari, Sonal b Motaghedi, Roja b Maclaren, Noel K b Adelaida Garcfa Gimeno, M c Sanz, Pascual d Rahier, Jacques e Rodrfguez Bada, Pablo f Cobo Vuilleumier, Nadia f Cuesta Muñoz, Antonio Luis f

a HADASSAH HEBREW UNIVERSITY MEDICAL CENTER (Israel)

b WEILL CORNELL MEDICAL COLLEGE (United States)

c CENTRO DE INVESTIGACIÓN BIOMÉDICA EN RED DE ENFERMEDADES RARAS CIBERER (Spain)

d CSIC (Spain)

e UNIVERSITÉ CATHOLIQUE DE LOUVAIN (Belgium)

f HOSPITAL REGIONAL UNIVERSITARIO DE MÁLAGA (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

ABC TRANSPORTER; ABC TRANSPORTER C8; ADENOSINE TRIPHOSPHATE SENSITIVE POTASSIUM CHANNEL; DIAZOXIDE; GLUCOKINASE; INSULIN; OCTREOTIDE; UNCLASSIFIED DRUG;

CASE REPORT; CELL PROLIFERATION; CHILD; CONTROLLED STUDY; FEMALE; GENE MUTATION; HUMAN; HUMAN TISSUE; HYPERINSULINEMIA; HYPOGLYCEMIA; INSULIN RELEASE; LETTER; PANCREAS ISLET; PANCREAS ISLET BETA CELL; PANCREAS RESECTION; PRESCHOOL CHILD; PRIORITY JOURNAL;

EID: 77950833891 PISSN: 00284793 EISSN: 15334406 Source Type: Journal
DOI: 10.1056/NEJMc0909845 Document Type: Letter

Times cited : (86)

References (5)

1
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- Beta-cell proliferation and apoptosis in the normal fetal and neonatal human pancreas and in persistent hyperinsulinemic hypoglycemia of infancy (PHHI)
- Kassem SA, Ariel I, Thornton PS, et al. Beta-cell proliferation and apoptosis in the normal fetal and neonatal human pancreas and in persistent hyperinsulinemic hypoglycemia of infancy (PHHI). Diabetes 2000;49:1325-1333
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- Kassem, S.A.¹ Ariel, I.² Thornton, P.S.³

2
- 0041319267
- Insights into the biochemical and genetic basis of glucokinase activation from naturally occurring hypoglycemia mutations
- DOI 10.2337/diabetes.52.9.2433
- 2. Gloyn AL, Noordam K, Willemsen MA, et al. Insights into the biochemical and genetic basis of glucokinase activation from naturally occurring hypoglycemia mutations. Diabetes 2003;52:2433-2440 (Pubitemid 37059517)
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- Gloyn, A.L.¹ Noordam, K.² Willemsen, M.A.A.P.³ Ellard, S.⁴ Lam, W.W.K.⁵ Campbell, I.W.⁶ Midgley, P.⁷ Shiota, C.⁸ Buettger, C.⁹ Magnuson, M.A.¹⁰ Matschinsky, F.M.¹¹ Hattersley, A.T.¹²

3
- 66649108506
- Extremes of clinical and enzymatic phenotypes in children with hyperinsulinism caused by glucokinase activating mutations
- Sayed S, Langdon DR., Odili S, et al. Extremes of clinical and enzymatic phenotypes in children with hyperinsulinism caused by glucokinase activating mutations. Diabetes 2009;58:1419-1427
- (2009) Diabetes , vol.58 , pp. 1419-1427
- Sayed, S.¹ Langdon, D.R.² Odili, S.³

4
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- Heterogeneity in disease severity in a family with a novel G68V GCK activating mutation causing persistent hyperinsulinaemic hypoglycaemia of infancy
- Wabitsch M, Lahr G, Van de Bunt M, et al. Heterogeneity in disease severity in a family with a novel G68V GCK activating mutation causing persistent hyperinsulinaemic hypoglycaemia of infancy. Diabet Med 2007;24:1393-1399
- (2007) Diabet Med , vol.24 , pp. 1393-1399
- Wabitsch, M.¹ Lahr, G.² Van De Bunt, M.³

5
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- Severe persistent hyperinsulinemic hypoglycemia due to a de novo glucokinase mutation
- Cuesta-Muñoz AL, Huopio H, Otonkoski T, et al. Severe persistent hyperinsulinemic hypoglycemia due to a de novo glucokinase mutation. Diabetes 2004;53:2164-2168
- (2004) Diabetes , vol.53 , pp. 2164-2168
- Cuesta-Muñoz, A.L.¹ Huopio, H.² Otonkoski, T.³

* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.