-
1
-
-
0030981722
-
Impaired mitochondrial function, oxidative stress and altered antioxidant enzyme activities following traumatic spinal cord injury
-
DOI 10.1016/S0006-8993(97)00573-8, PII S0006899397005738
-
Azbill RD, Mu X, Bruce-Keller AJ, Mattson MP, Springer JE. Impaired mitochondrial function, oxidative stress and altered antioxidant enzyme activities following traumatic spinal cord injury. Brain Res 1997; 765: 283-290. (Pubitemid 27397624)
-
(1997)
Brain Research
, vol.765
, Issue.2
, pp. 283-290
-
-
Azbill, R.D.1
Mu, X.2
Bruce-Keller, A.J.3
Mattson, M.P.4
Springer, J.E.5
-
2
-
-
0035169773
-
Promoting glutathione synthesis after spinal cord trauma decreases secondary damage and promotes retention of function
-
DOI 10.1096/fj.00-0228com
-
Kamencic H, Griebel RW, Lyon AW, Paterson PG, Juurlink BH. Promoting glutathione synthesis after spinal cord trauma decreases secondary damage and promotes retention of function. FASEB J 2001; 15: 243-250. (Pubitemid 32061675)
-
(2001)
FASEB Journal
, vol.15
, Issue.1
, pp. 243-250
-
-
Kamencic, H.1
Griebel, R.W.2
Lyon, A.W.3
Paterson, P.G.4
Juurlink, B.H.J.5
-
3
-
-
0344009549
-
NAD(P)H oxidase, superoxide dismutase, catalase, glutathione peroxidase and nitric oxide synthase expression in subacute spinal cord injury
-
Vaziri ND, Lee YS, Lin CY, Lin VW, Sindhu RK. NAD(P)H oxidase, superoxide dismutase, catalase, glutathione peroxidase and nitric oxide synthase expression in subacute spinal cord injury. Brain Res 2004; 995: 76-83.
-
(2004)
Brain Res
, vol.995
, pp. 76-83
-
-
Vaziri, N.D.1
Lee, Y.S.2
Lin, C.Y.3
Lin, V.W.4
Sindhu, R.K.5
-
4
-
-
33646578441
-
Overexpression of SOD1 in transgenic rats attenuates nuclear translocation of endonuclease G and apoptosis after spinal cord injury
-
Yu F, Sugawara T, Nishi T, Liu J, Chan PH. Overexpression of SOD1 in transgenic rats attenuates nuclear translocation of endonuclease G and apoptosis after spinal cord injury. J Neurotrauma 2006; 23: 595-603. (Pubitemid 43728049)
-
(2006)
Journal of Neurotrauma
, vol.23
, Issue.5
, pp. 595-603
-
-
Yu, F.1
Sugawara, T.2
Nishi, T.3
Liu, J.4
Chan, P.H.5
-
5
-
-
33644520285
-
NMDA receptors mediate calcium accumulation in myelin during chemical ischaemia
-
DOI 10.1038/nature04474, PII N04474
-
Micu I, Jiang Q, Coderre E, Ridsdale A, Zhang L, Woulfe J et al. NMDA receptors mediate calcium accumulation in myelin during chemical ischaemia. Nature 2006; 439: 988-992. (Pubitemid 43292418)
-
(2006)
Nature
, vol.439
, Issue.7079
, pp. 988-992
-
-
Micu, I.1
Jiang, Q.2
Coderre, E.3
Ridsdale, A.4
Zhang, L.5
Woulfe, J.6
Yin, X.7
Trapp, B.D.8
McRory, J.E.9
Rehak, R.10
Zamponi, G.W.11
Wang, W.12
Stys, P.K.13
-
6
-
-
0029130550
-
Glutamate release and free radical production following brain injury: Effects of posttraumatic hypothermia
-
Globus MY, Alonso O, Dietrich WD, Busto R, Ginsberg MD. Glutamate release and free radical production following brain injury: effects of posttraumatic hypothermia. J Neurochem 1995; 65: 1704-1711.
-
(1995)
J Neurochem
, vol.65
, pp. 1704-1711
-
-
Globus, M.Y.1
Alonso, O.2
Dietrich, W.D.3
Busto, R.4
Ginsberg, M.D.5
-
7
-
-
0031020308
-
Role of NMDA and non-NMDA ionotropic glutamate receptors in traumatic spinal cord axonal injury
-
Agrawal SK, Fehlings MG. Role of NMDA and non-NMDA ionotropic glutamate receptors in traumatic spinal cord axonal injury. J Neurosci 1997; 17: 1055-1063. (Pubitemid 27043964)
-
(1997)
Journal of Neuroscience
, vol.17
, Issue.3
, pp. 1055-1063
-
-
Agrawal, S.K.1
Fehlings, M.G.2
-
8
-
-
26944436689
-
Protective use of N-methyl-D-aspartate receptor antagonists as a spinoplegia against excitatory amino acid neurotoxicity
-
DOI 10.1016/j.jvs.2005.05.052, PII S0741521405009663
-
Cho Y, Ueda T, Mori A, Shimizu H, Haga Y, Yozu R. Protective use of N-methyl-D-aspartate receptor antagonists as a spinoplegia against excitatory amino acid neurotoxicity. J Vasc Surg 2005; 42: 765-771. (Pubitemid 41483505)
-
(2005)
Journal of Vascular Surgery
, vol.42
, Issue.4
, pp. 765-771
-
-
Cho, Y.1
Ueda, T.2
Mori, A.3
Shimizu, H.4
Haga, Y.5
Yozu, R.6
-
9
-
-
0030474894
-
Pretreatment with NMDA receptor antagonist MK801 improves neurophysiological outcome after an acute spinal cord injury
-
Haghighi SS, Johnson GC, de Vergel CF, Vergel Rivas BJ. Pretreatment with NMDA receptor antagonist MK801 improves neurophysiological outcome after an acute spinal cord injury. Neurol Res 1996; 18: 509-515.
-
(1996)
Neurol Res
, vol.18
, pp. 509-515
-
-
Haghighi, S.S.1
Johnson, G.C.2
De Vergel, C.F.3
Vergel Rivas, B.J.4
-
10
-
-
0032698797
-
Apoptosis following spinal cord injury in rats and preventative effect of N-methyl-D-aspartate receptor antagonist
-
Wada S, Yone K, Ishidou Y, Nagamine T, Nakahara S, Niiyama T et al. Apoptosis following spinal cord injury in rats and preventative effect of N-methyl-D-aspartate receptor antagonist. J Neurosurg 1999; 91 (1 Suppl): 98-104.
-
(1999)
J Neurosurg
, vol.91
, Issue.1 SUPPL.
, pp. 98-104
-
-
Wada, S.1
Yone, K.2
Ishidou, Y.3
Nagamine, T.4
Nakahara, S.5
Niiyama, T.6
-
11
-
-
0342813092
-
Delayed antagonism of AMPA/kainate receptors reduces long-term functional deficits resulting from spinal cord trauma
-
DOI 10.1006/exnr.1997.6506
-
Wrathall JR, Teng YD, Marriott R. Delayed antagonism of AMPA/kainate receptors reduces long-term functional deficits resulting from spinal cord trauma. Exp Neurol 1997; 145 (2 Pt 1): 565-573. (Pubitemid 27314807)
-
(1997)
Experimental Neurology
, vol.145
, Issue.2
, pp. 565-573
-
-
Wrathall, J.R.1
Teng, Y.D.2
Marriott, R.3
-
12
-
-
0036332542
-
Blockade of NMDA-receptors or calcium-channels attenuates the ischaemia-evoked efflux of glutamate and phosphoethanolamine and depression of neuronal activity in rat organotypic hippocampal slice cultures
-
DOI 10.1016/S1631-0691(02)01451-8
-
Robert F, Bert L, Stoppini L. Blockade of NMDA-receptors or calcium-channels attenuates the ischaemia-evoked efflux of glutamate and phosphoethanolamine and depression of neuronal activity in rat organotypic hippocampal slice cultures. C R Biol 2002; 325: 495-504. (Pubitemid 34855248)
-
(2002)
Comptes Rendus - Biologies
, vol.325
, Issue.4
, pp. 495-504
-
-
Robert, F.1
Bert, L.2
Stoppini, L.3
-
13
-
-
0022607625
-
Experimental spinal cord injury: Quantitation of axonal damage by automated image analysis
-
Iizuka H, Yamamoto T, Iwasaki Y, Konno H, Kadoya S. Experimental spinal cord injury: quantification of axonal damage by automated image analysis. J Neurosurg 1986; 64: 304-308. (Pubitemid 16140347)
-
(1986)
Journal of Neurosurgery
, vol.64
, Issue.2
, pp. 304-308
-
-
Iizuka, H.1
Yamamoto, T.2
Iwasaki, Y.3
-
15
-
-
77049306406
-
The glutathione instability of drug-sensitive red cells; A new method for the in vitro detection of drug sensitivity
-
Beutler E. The glutathione instability of drug-sensitive red cells; a new method for the in vitro detection of drug sensitivity. J Lab Clin Med 1957; 49: 84-95.
-
(1957)
J Lab Clin Med
, vol.49
, pp. 84-95
-
-
Beutler, E.1
-
16
-
-
0018384650
-
Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction
-
DOI 10.1016/0003-2697(79)90738-3
-
Ohkawa H, Ohishi N, Yagi K. Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction. Anal Biochem 1979; 95: 351-358. (Pubitemid 9194347)
-
(1979)
Analytical Biochemistry
, vol.95
, Issue.2
, pp. 351-358
-
-
Ohkawa, H.1
Ohishi, N.2
Yagi, K.3
-
18
-
-
4244087065
-
Reactive oxygen species in living systems: Source, biochemistry, and role in human disease
-
Halliwell B. Reactive oxygen species in living systems: source, biochemistry, and role in human disease. Am J Med 1991; 91 (3C): 14S-22S.
-
(1991)
Am J Med
, vol.91
, Issue.3 C
-
-
Halliwell, B.1
-
19
-
-
0036904245
-
Recent advances in pathophysiology and treatment of spinal cord injury
-
Hulsebosch CE. Recent advances in pathophysiology and treatment of spinal cord injury. Adv Physiol Educ 2002; 26: 238-255.
-
(2002)
Adv Physiol Educ
, vol.26
, pp. 238-255
-
-
Hulsebosch, C.E.1
-
20
-
-
0036676084
-
NBQX treatment improves mitochondrial function and reduces oxidative events after spinal cord injury
-
Mu X, Azbill RD, Springer JE. NBQX treatment improves mitochondrial function and reduces oxidative events after spinal cord injury. J Neurotrauma 2002; 19: 917-927.
-
(2002)
J Neurotrauma
, vol.19
, pp. 917-927
-
-
Mu, X.1
Azbill, R.D.2
Springer, J.E.3
|