Dipyridamole increases gap junction coupling in bovine GM-7373 aortic endothelial cells by a cAMP-protein kinase A dependent pathway

Journal of Bioenergetics and Biomembranes

Volumn 42, Issue 1, 2010, Pages 79-84

  Begandt, D ^a   Bintig, W ^a   Oberheide, K ^a   Schlie, S ^a   Ngezahayo, A ^a  

^a LEIBNIZ UNIVERSITY HANNOVER   (Germany)

Author keywords

cAMP; Connexins; Dipyridamole; Endothelial cells; Gap junction; H 89; PKA; Scrape loading

Indexed keywords

8 BROMO CYCLIC AMP; CYCLIC AMP DEPENDENT PROTEIN KINASE; CYCLIC GMP; DIPYRIDAMOLE; FORSKOLIN; N [2 (4 BROMOCINNAMYLAMINO)ETHYL] 5 ISOQUINOLINESULFONAMIDE;

ANIMAL CELL; ARTERY ENDOTHELIUM; ARTICLE; CONCENTRATION RESPONSE; CONTROLLED STUDY; COW; DRUG ANTAGONISM; DRUG MECHANISM; ENDOTHELIUM CELL; ENZYME ACTIVITY; ENZYME INHIBITION; ENZYME PHOSPHORYLATION; GAP JUNCTION; NONHUMAN;

ANIMALS; CATTLE; CELL LINE; CONNEXINS; CYCLIC AMP-DEPENDENT PROTEIN KINASES; DIPYRIDAMOLE; ENDOTHELIAL CELLS; FIBRINOLYTIC AGENTS; GAP JUNCTIONS; ISOQUINOLINES; MODELS, BIOLOGICAL; PROTEIN KINASE INHIBITORS; SULFONAMIDES;

BOVINAE;

EID: 77950021111     PISSN: 0145479X     EISSN: 15736881     Source Type: Journal    
DOI: 10.1007/s10863-009-9262-2     Document Type: Article

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