EBNA2 is required for protection of latently epstein-barr virus-infected B cells against specific apoptotic stimuli

Journal of Virology

Volumn 78, Issue 22, 2004, Pages 12694-12697

  Jae, Myun Lee ^a   Lee, Kyoung Ho ^b,f   Farrell, Christopher J ^c   Ling, Paul D ^d   Kempkes, Bettina ^e   Jeon, Han Park ^a   Hayward, S Diane ^b,c  

^a Yonsei University   (South Korea)

^b JOHNS HOPKINS UNIVERSITY   (United States)

^c JOHNS HOPKINS UNIVERSITY   (United States)

^d BAYLOR COLLEGE OF MEDICINE   (United States)

^e INSTITUTE OF EPIDEMIOLOGY   (Germany)

^f Yonsei University Wonju   (South Korea)

Author keywords

[No Author keywords available]

Indexed keywords

EPSTEIN BARR VIRUS ANTIGEN; EPSTEIN BARR VIRUS ANTIGEN 2; ESTROGEN; ETOPOSIDE; FLUOROURACIL; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; B LYMPHOCYTE; CELL PROTECTION; EPSTEIN BARR VIRUS; GENE DELETION; GENE EXPRESSION; PRIORITY JOURNAL; VIRUS INFECTION;

APOPTOSIS; B-LYMPHOCYTES; DNA-BINDING PROTEINS; EPSTEIN-BARR VIRUS NUCLEAR ANTIGENS; HERPESVIRUS 4, HUMAN; RECEPTORS, CYTOPLASMIC AND NUCLEAR; RECEPTORS, STEROID; TRANSCRIPTION FACTORS; TUMOR NECROSIS FACTOR-ALPHA; VIRAL MATRIX PROTEINS; VIRAL PROTEINS; VIRUS LATENCY;

EID: 7644227541     PISSN: 0022538X     EISSN: None     Source Type: Journal    
DOI: 10.1128/JVI.78.22.12694-12697.2004     Document Type: Article

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