Cardiac Ito, KCNE2, and Brugada syndrome: Promiscuous subunit interactions, or what happens in HEK cells stays in HEK cells?

Heart Rhythm

Volumn 7, Issue 2, 2010, Pages 206-207

  Tomaselli, Gordon F ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ION CHANNEL; POTASSIUM CHANNEL KCNE2; POTASSIUM CHANNEL KCNQ1; UNCLASSIFIED DRUG; VOLTAGE GATED POTASSIUM CHANNEL KV4.3;

BRUGADA SYNDROME; COMPLEX FORMATION; CONTROLLED STUDY; DOWN REGULATION; EDITORIAL; ELECTROCARDIOGRAPHY; ELECTROPHYSIOLOGY; GENE EXPRESSION; GENE MUTATION; GENETIC PREDISPOSITION; HEART ARRHYTHMIA; HUMAN; HUMAN CELL; ION CURRENT; LOSS OF FUNCTION MUTATION; PHENOTYPE; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; QT PROLONGATION;

ARRHYTHMIAS, CARDIAC; BRUGADA SYNDROME; CELL LINE; CELLS, CULTURED; CLONING, MOLECULAR; DEATH, SUDDEN, CARDIAC; ELECTROPHYSIOLOGY; GENETIC VECTORS; HEART CONDUCTION SYSTEM; HUMANS; KINETICS; KV CHANNEL-INTERACTING PROTEINS; MEMBRANE POTENTIALS; MUTATION; PATCH-CLAMP TECHNIQUES; PHENOTYPE; POTASSIUM CHANNELS, VOLTAGE-GATED; SHAL POTASSIUM CHANNELS; TRANSFECTION;

EID: 74449086930     PISSN: 15475271     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.hrthm.2009.10.029     Document Type: Editorial

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