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Volumn 55, Issue 1, 2010, Pages 23-25

Mitogen-activated protein kinases as biomarkers of hypertension or cardiac pressure overload

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; MITOGEN ACTIVATED PROTEIN KINASE 7; MITOGEN ACTIVATED PROTEIN KINASE P38; PROTEIN C JUN;

EID: 73849139311     PISSN: 0194911X     EISSN: None     Source Type: Journal    
DOI: 10.1161/HYPERTENSIONAHA.109.141960     Document Type: Note
Times cited : (11)

References (7)
  • 2
    • 53449097061 scopus 로고    scopus 로고
    • MAPK signalling in cardiovascular health and disease: Molecular mechanisms and therapeutic targets
    • Muslin AJ. MAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets. Clin Sci. 2008;115: 203-218.
    • (2008) Clin Sci , vol.115 , pp. 203-218
    • Muslin, A.J.1
  • 3
    • 34548695952 scopus 로고    scopus 로고
    • Mitogen-activated protein kinases in heart development and diseases
    • Wang Y. Mitogen-activated protein kinases in heart development and diseases. Circulation. 2007;116:1413-1423.
    • (2007) Circulation , vol.116 , pp. 1413-1423
    • Wang, Y.1
  • 4
    • 0036843158 scopus 로고    scopus 로고
    • Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death
    • Bueno OF, Molkentin JD. Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death. Circ Res. 2002;91: 776-781.
    • (2002) Circ Res , vol.91 , pp. 776-781
    • Bueno, O.F.1    Molkentin, J.D.2
  • 5
    • 33748323383 scopus 로고    scopus 로고
    • Regulation of cardiac hypertrophy by intra-cellular signalling pathways
    • Heineke J, Molkentin JD. Regulation of cardiac hypertrophy by intra-cellular signalling pathways. Nat Rev Mol Cell Biol. 2006;7:589-600.
    • (2006) Nat Rev Mol Cell Biol , vol.7 , pp. 589-600
    • Heineke, J.1    Molkentin, J.D.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.