-
1
-
-
61449157818
-
Disrupted serotonergic and sympathoadrenal systems in patients with chronic heart failure may serve as new therapeutic targets and novel biomarkers to assess severity, progression and response to treatment
-
Nigmatullina R.R., Kirillova V.V., Jourjikiya R.K., Mukhamedyarov M.A., Kudrin V.S., Klodt P.M., and Palotas A. Disrupted serotonergic and sympathoadrenal systems in patients with chronic heart failure may serve as new therapeutic targets and novel biomarkers to assess severity, progression and response to treatment. Cardiology 113 (2009) 277-286
-
(2009)
Cardiology
, vol.113
, pp. 277-286
-
-
Nigmatullina, R.R.1
Kirillova, V.V.2
Jourjikiya, R.K.3
Mukhamedyarov, M.A.4
Kudrin, V.S.5
Klodt, P.M.6
Palotas, A.7
-
2
-
-
0028475705
-
Serotonergic mechanisms in heart failure
-
Chandra M., Gupta V., Johri A.K., Misra R., Kumar A., Gujrati V., and Shanker K. Serotonergic mechanisms in heart failure. Indian Heart J. 46 (1994) 153-156
-
(1994)
Indian Heart J.
, vol.46
, pp. 153-156
-
-
Chandra, M.1
Gupta, V.2
Johri, A.K.3
Misra, R.4
Kumar, A.5
Gujrati, V.6
Shanker, K.7
-
3
-
-
36448954656
-
Serotonin responsiveness through 5-HT2A and 5-HT4 receptors is differentially regulated in hypertrophic and failing rat cardiac ventricle
-
Brattelid T., Qvigstad E., Birkeland J.A., Swift F., Bekkevold S.V., Krobert K.A., Sejersted O.M., Skomedal T., Osnes J.B., Levy F.O., and Sjaastad I. Serotonin responsiveness through 5-HT2A and 5-HT4 receptors is differentially regulated in hypertrophic and failing rat cardiac ventricle. J. Mol. Cell. Cardiol. 43 (2007) 767-779
-
(2007)
J. Mol. Cell. Cardiol.
, vol.43
, pp. 767-779
-
-
Brattelid, T.1
Qvigstad, E.2
Birkeland, J.A.3
Swift, F.4
Bekkevold, S.V.5
Krobert, K.A.6
Sejersted, O.M.7
Skomedal, T.8
Osnes, J.B.9
Levy, F.O.10
Sjaastad, I.11
-
4
-
-
0025236458
-
Effects of ketanserin on left ventricular hypertrophy in hypertensive patients
-
Cobo C., Alcocer L., and Chavez A. Effects of ketanserin on left ventricular hypertrophy in hypertensive patients. Cardiovasc. Drugs Ther. 4 Suppl. 1 (1990) 73-76
-
(1990)
Cardiovasc. Drugs Ther.
, vol.4
, Issue.SUPPL. 1
, pp. 73-76
-
-
Cobo, C.1
Alcocer, L.2
Chavez, A.3
-
5
-
-
61749092853
-
Genetic deletion of MAO-A promotes serotonin-dependent ventricular hypertrophy by pressure overload
-
Lairez O., Calise D., Bianchi P., Ordener C., Spreux-Varoquaux O., Guilbeau-Frugier C., Escourrou G., Seif I., Roncalli J., Pizzinat N., Galinier M., Parini A., and Mialet-Perez J. Genetic deletion of MAO-A promotes serotonin-dependent ventricular hypertrophy by pressure overload. J. Mol. Cell. Cardiol. 46 (2009) 587-595
-
(2009)
J. Mol. Cell. Cardiol.
, vol.46
, pp. 587-595
-
-
Lairez, O.1
Calise, D.2
Bianchi, P.3
Ordener, C.4
Spreux-Varoquaux, O.5
Guilbeau-Frugier, C.6
Escourrou, G.7
Seif, I.8
Roncalli, J.9
Pizzinat, N.10
Galinier, M.11
Parini, A.12
Mialet-Perez, J.13
-
6
-
-
68049115746
-
Dose-dependent activation of distinct hypertrophic pathways by serotonin in cardiac cells
-
Villeneuve C., Caudrillier A., Ordener C., Pizzinat N., Parini A., and Mialet-Perez J. Dose-dependent activation of distinct hypertrophic pathways by serotonin in cardiac cells. Am. J. Physiol. Heart Circ. Physiol. 297 (2009) H821-H828
-
(2009)
Am. J. Physiol. Heart Circ. Physiol.
, vol.297
-
-
Villeneuve, C.1
Caudrillier, A.2
Ordener, C.3
Pizzinat, N.4
Parini, A.5
Mialet-Perez, J.6
-
7
-
-
1542297759
-
A small molecular activator of cardiac hypertrophy uncovered in a chemical screen for modifiers of the calcineurin signaling pathway
-
Bush E., Fielitz J., Melvin L., Martinez-Arnold M., McKinsey T.A., Plichta R., and Olson E.N. A small molecular activator of cardiac hypertrophy uncovered in a chemical screen for modifiers of the calcineurin signaling pathway. Proc. Natl. Acad. Sci. USA 101 (2004) 2870-2875
-
(2004)
Proc. Natl. Acad. Sci. USA
, vol.101
, pp. 2870-2875
-
-
Bush, E.1
Fielitz, J.2
Melvin, L.3
Martinez-Arnold, M.4
McKinsey, T.A.5
Plichta, R.6
Olson, E.N.7
-
8
-
-
0347320747
-
Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy
-
Wilkins B.J., Dai Y.S., Bueno O.F., Parsons S.A., Xu J., Plank D.M., Jones F., Kimball T.R., and Molkentin J.D. Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy. Circ. Res. 94 (2004) 110-118
-
(2004)
Circ. Res.
, vol.94
, pp. 110-118
-
-
Wilkins, B.J.1
Dai, Y.S.2
Bueno, O.F.3
Parsons, S.A.4
Xu, J.5
Plank, D.M.6
Jones, F.7
Kimball, T.R.8
Molkentin, J.D.9
-
9
-
-
44649128517
-
TRP channel and cardiovascular disease
-
Watanabe H., Murakami M., Ohba T., Takahashi Y., and Ito H. TRP channel and cardiovascular disease. Pharmacol. Ther. 118 (2008) 337-351
-
(2008)
Pharmacol. Ther.
, vol.118
, pp. 337-351
-
-
Watanabe, H.1
Murakami, M.2
Ohba, T.3
Takahashi, Y.4
Ito, H.5
-
10
-
-
22644442369
-
Emerging functions of 10 types of TRP cationic channel in vascular smooth muscle
-
Beech D.J. Emerging functions of 10 types of TRP cationic channel in vascular smooth muscle. Clin. Exp. Pharmacol. Physiol. 32 (2005) 597-603
-
(2005)
Clin. Exp. Pharmacol. Physiol.
, vol.32
, pp. 597-603
-
-
Beech, D.J.1
-
11
-
-
33845597014
-
Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling
-
Bush E.W., Hood D.B., Papst P.J., Chapo J.A., Minobe W., Bristow M.R., Olson E.N., and McKinsey T.A. Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling. J. Biol. Chem. 281 (2006) 33487-33496
-
(2006)
J. Biol. Chem.
, vol.281
, pp. 33487-33496
-
-
Bush, E.W.1
Hood, D.B.2
Papst, P.J.3
Chapo, J.A.4
Minobe, W.5
Bristow, M.R.6
Olson, E.N.7
McKinsey, T.A.8
-
12
-
-
33749338744
-
Calcineurin-dependent cardiomyopathy is activated by TRPC in the adult mouse heart
-
Nakayama H., Wilkin B.J., Bodi I., and Molkentin J.D. Calcineurin-dependent cardiomyopathy is activated by TRPC in the adult mouse heart. FASEB J. 20 (2006) 1660-1670
-
(2006)
FASEB J.
, vol.20
, pp. 1660-1670
-
-
Nakayama, H.1
Wilkin, B.J.2
Bodi, I.3
Molkentin, J.D.4
-
13
-
-
33751104984
-
TRPC3 and TRPC6 are essential for angiotensin II-induced cardiac hypertrophy
-
Onohara N., Nishida M., Inoue R., Kobayashi H., Sumimoto H., Sato Y., Mori Y., Nagao T., and Kurose H. TRPC3 and TRPC6 are essential for angiotensin II-induced cardiac hypertrophy. EMBO J. 25 (2006) 5305-5316
-
(2006)
EMBO J.
, vol.25
, pp. 5305-5316
-
-
Onohara, N.1
Nishida, M.2
Inoue, R.3
Kobayashi, H.4
Sumimoto, H.5
Sato, Y.6
Mori, Y.7
Nagao, T.8
Kurose, H.9
-
14
-
-
33845317604
-
TRPC6 fulfills a calcineurin signaling circuit during pathologic cardiac remodeling
-
Kuwahara K., Wang Y., McAnally J., Richardson J.A., Bassel-Duby R., Hill J.A., and Olson E.N. TRPC6 fulfills a calcineurin signaling circuit during pathologic cardiac remodeling. J. Clin. Invest. 116 (2006) 3114-3126
-
(2006)
J. Clin. Invest.
, vol.116
, pp. 3114-3126
-
-
Kuwahara, K.1
Wang, Y.2
McAnally, J.3
Richardson, J.A.4
Bassel-Duby, R.5
Hill, J.A.6
Olson, E.N.7
-
15
-
-
33847372100
-
Upregulation of TRPC1 in the development of cardiac hypertrophy
-
Ohba T., Watanabe H., Murakami M., Takahashi Y., Iino K., Kuromitsu S., Mori Y., Ono K., Iijima T., and Ito H. Upregulation of TRPC1 in the development of cardiac hypertrophy. J. Mol. Cell. Cardiol. 42 (2007) 498-507
-
(2007)
J. Mol. Cell. Cardiol.
, vol.42
, pp. 498-507
-
-
Ohba, T.1
Watanabe, H.2
Murakami, M.3
Takahashi, Y.4
Iino, K.5
Kuromitsu, S.6
Mori, Y.7
Ono, K.8
Iijima, T.9
Ito, H.10
-
16
-
-
11144313355
-
Urotensin-II-mediated cardiomyocyte hypertrophy: effect of receptor antagonism and role of inflammatory mediators
-
Johns D.G., Ao Z., Naselsky D., Herold C.L., Maniscalco K., Sarov-Blat L., Steplewski K., Aiyar N., and Douglas S.A. Urotensin-II-mediated cardiomyocyte hypertrophy: effect of receptor antagonism and role of inflammatory mediators. Naunyn Schmiedebergs Arch. Pharmacol. 370 (2004) 238-250
-
(2004)
Naunyn Schmiedebergs Arch. Pharmacol.
, vol.370
, pp. 238-250
-
-
Johns, D.G.1
Ao, Z.2
Naselsky, D.3
Herold, C.L.4
Maniscalco, K.5
Sarov-Blat, L.6
Steplewski, K.7
Aiyar, N.8
Douglas, S.A.9
-
17
-
-
57749083482
-
Metabolic switch and hypertrophy of cardiomyocytes following treatment with angiotensin II are prevented by AMP-activated protein kinase
-
Stuck B.J., Lenski M., Bohm M., and Laufs U. Metabolic switch and hypertrophy of cardiomyocytes following treatment with angiotensin II are prevented by AMP-activated protein kinase. J. Biol. Chem. 283 (2008) 32562-32569
-
(2008)
J. Biol. Chem.
, vol.283
, pp. 32562-32569
-
-
Stuck, B.J.1
Lenski, M.2
Bohm, M.3
Laufs, U.4
-
18
-
-
51749116707
-
Roles of TRP channels in the development of cardiac hypertrophy
-
Nishida M., and Kurose H. Roles of TRP channels in the development of cardiac hypertrophy. Naunyn Schmiedebergs Arch. Pharmacol. 378 (2008) 395-406
-
(2008)
Naunyn Schmiedebergs Arch. Pharmacol.
, vol.378
, pp. 395-406
-
-
Nishida, M.1
Kurose, H.2
-
19
-
-
16344378609
-
A new hypertrophic mechanism of serotonin in cardiac myocytes: receptor-independent ROS generation
-
Bianchi P., Pimentel D.R., Murphy M.P., Colucci W.S., and Parini A. A new hypertrophic mechanism of serotonin in cardiac myocytes: receptor-independent ROS generation. FASEB J. 19 (2005) 641-643
-
(2005)
FASEB J.
, vol.19
, pp. 641-643
-
-
Bianchi, P.1
Pimentel, D.R.2
Murphy, M.P.3
Colucci, W.S.4
Parini, A.5
-
20
-
-
72449170101
-
TRPC1 channels are critical for hypertrophic signaling in the heart
-
Seth M., Zhang Z.S., Mao L., Graham V., Burch J., Stiber J., Tsiokas L., Winn M., Abramowitz J., Rockman H.A., Birnbaumer L., and Rosenberg P. TRPC1 channels are critical for hypertrophic signaling in the heart. Circ. Res. 105 (2009) 1023-1030
-
(2009)
Circ. Res.
, vol.105
, pp. 1023-1030
-
-
Seth, M.1
Zhang, Z.S.2
Mao, L.3
Graham, V.4
Burch, J.5
Stiber, J.6
Tsiokas, L.7
Winn, M.8
Abramowitz, J.9
Rockman, H.A.10
Birnbaumer, L.11
Rosenberg, P.12
-
21
-
-
49849087015
-
Diacylglycerol kinase-epsilon restores cardiac dysfunction under chronic pressure overload: a new specific regulator of Galpha(q) signaling cascade
-
Niizeki T., Takeishi Y., Kitahara T., Arimoto T., Ishino M., Bilim O., Suzuki S., Sasaki T., Nakajima O., Walsh R.A., Goto K., and Kubota I. Diacylglycerol kinase-epsilon restores cardiac dysfunction under chronic pressure overload: a new specific regulator of Galpha(q) signaling cascade. Am. J. Physiol. Heart Circ. Physiol. 295 (2008) H245-H255
-
(2008)
Am. J. Physiol. Heart Circ. Physiol.
, vol.295
-
-
Niizeki, T.1
Takeishi, Y.2
Kitahara, T.3
Arimoto, T.4
Ishino, M.5
Bilim, O.6
Suzuki, S.7
Sasaki, T.8
Nakajima, O.9
Walsh, R.A.10
Goto, K.11
Kubota, I.12
-
22
-
-
0037188517
-
Subunit composition of mammalian transient receptor potential channels in living cells
-
Hofmann T., Schaefer M., Schultz G., and Gudermann T. Subunit composition of mammalian transient receptor potential channels in living cells. Proc. Natl. Acad. Sci. USA 99 (2002) 7461-7466
-
(2002)
Proc. Natl. Acad. Sci. USA
, vol.99
, pp. 7461-7466
-
-
Hofmann, T.1
Schaefer, M.2
Schultz, G.3
Gudermann, T.4
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