Vaccinia-induced epidermal growth factor receptor-MEK signalling and the anti-apoptotic protein F1L synergize to suppress cell death during infection

Cellular Microbiology

Volumn 11, Issue 8, 2009, Pages 1208-1218

  Postigo, Antonio ^a   Martin, Morag C ^a   Dodding, Mark P ^a   Way, Michael ^a  

^a IMPERIAL CANCER RESEARCH FUND   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

BH3 PROTEIN; CASPASE 3; EPIDERMAL GROWTH FACTOR RECEPTOR; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN BAD; PROTEIN BCL 2; PROTEIN F1L; UNCLASSIFIED DRUG; VIRULENCE FACTOR; VIRUS PROTEIN;

APOPTOSIS; ARTICLE; CELL DEATH; CELL PROTECTION; CELL SURVIVAL; CONTROLLED STUDY; GENE ACTIVATION; GENE EXPRESSION; HUMAN; HUMAN CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN CLEAVAGE; VACCINIA VIRUS; VIRUS CELL INTERACTION; VIRUS VIRULENCE;

ANTIMETABOLITES, ANTINEOPLASTIC; APOPTOSIS; BCL-ASSOCIATED DEATH PROTEIN; CYTARABINE; EPIDERMAL GROWTH FACTOR; GENE EXPRESSION REGULATION, VIRAL; HELA CELLS; HOST-PATHOGEN INTERACTIONS; HUMANS; MAP KINASE SIGNALING SYSTEM; PEPTIDES; VACCINIA; VACCINIA VIRUS; VIRAL PROTEINS; VIRULENCE; VIRUS REPLICATION;

EID: 67650102351     PISSN: 14625814     EISSN: 14625822     Source Type: Journal    
DOI: 10.1111/j.1462-5822.2009.01327.x     Document Type: Article

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