|
|
Volumn 8, Issue 12, 2009, Pages 1813-1815
|
|
TGFβ versatility: PI3K as a critical mediator of distinct cell type and context specific responses
|
|
Author keywords
EMT; Epithelia; Fibroblasts; Non Smad; PI3K; Signaling; Smad; TGF
|
|
Indexed keywords
ABELSON KINASE;
CYCLIN DEPENDENT KINASE INHIBITOR;
IMATINIB;
MAMMALIAN TARGET OF RAPAMYCIN;
MITOGEN ACTIVATED PROTEIN KINASE;
P21 ACTIVATED KINASE 2;
PHOSPHATIDYLINOSITOL 3 KINASE;
PHOSPHOPROTEIN PHOSPHATASE 2A;
PROTEIN KINASE B;
PROTEINASE ACTIVATED RECEPTOR;
PROTEINASE ACTIVATED RECEPTOR 6;
SMAD PROTEIN;
TRANSCRIPTION FACTOR FOXO;
TRANSFORMING GROWTH FACTOR BETA;
UNCLASSIFIED DRUG;
PLATELET DERIVED GROWTH FACTOR B;
PROTEIN KINASE INHIBITOR;
ANGIOGENESIS;
CELL CYCLE ARREST;
CELL DIFFERENTIATION;
CELL GROWTH;
CELL MIGRATION;
CELL STRUCTURE;
EDITORIAL;
EPITHELIUM CELL;
FIBROBLAST;
GENE EXPRESSION;
LUNG FIBROSIS;
MESENCHYME CELL;
NONHUMAN;
PLEIOTROPY;
PROTEIN FUNCTION;
PROTEIN PHOSPHORYLATION;
SIGNAL TRANSDUCTION;
URETER OBSTRUCTION;
ANIMAL;
HUMAN;
METABOLISM;
1-PHOSPHATIDYLINOSITOL 3-KINASE;
ANIMALS;
HUMANS;
PROTEIN KINASE INHIBITORS;
PROTO-ONCOGENE PROTEINS C-SIS;
SIGNAL TRANSDUCTION;
SMAD PROTEINS;
TRANSFORMING GROWTH FACTOR BETA;
|
|
EID: 67649329386
PISSN: 15384101
EISSN: 15514005
Source Type: Journal
DOI: 10.4161/cc.8.12.8828 Document Type: Editorial |
|
Times cited : (4)
|
|
References (14)
|
