-
1
-
-
33646161959
-
The MLL recombinome of acute leukemias
-
Meyer C, Schneider B, Jakob S, et al. The MLL recombinome of acute leukemias. Leukemia. 2006;20:777-784.
-
(2006)
Leukemia
, vol.20
, pp. 777-784
-
-
Meyer, C.1
Schneider, B.2
Jakob, S.3
-
2
-
-
33947421739
-
Why and how to quantify minimal residual disease in acute lymphoblastic leukemia?
-
Szczepański T. Why and how to quantify minimal residual disease in acute lymphoblastic leukemia? Leukemia. 2007;21:622-626.
-
(2007)
Leukemia
, vol.21
, pp. 622-626
-
-
Szczepański, T.1
-
3
-
-
33644512771
-
Monitoring minimal residual disease by quantification of genomic chromosomal breakpoint sequences in acute leukemias with MLL aberrations
-
DOI 10.1038/sj.leu.2404082, PII 2404082
-
Burmeister T, Marschalek R, Schneider B, et al. Monitoring minimal residual disease by quantification of genomic chromosomal breakpoint sequences in acute leukemias with MLL aberrations. Leukemia. 2006;20:451-457. (Pubitemid 43291743)
-
(2006)
Leukemia
, vol.20
, Issue.3
, pp. 451-457
-
-
Burmeister, T.1
Marschalek, R.2
Schneider, B.3
Meyer, C.4
Gokbuget, N.5
Schwartz, S.6
Hoelzer, D.7
Thiel, E.8
-
4
-
-
0032530350
-
Immunophenotypic and genotypic features, clinical characteristics and treatment outcome of adult pro-B acute lymphoblastic leukemia: Results of the German multicenter trials GMALL 03/87 and 04/89
-
Ludwig WD, Rieder H, Bartram CR, et al. Immunophenotypic and genotypic features, clinical characteristics, and treatment outcome of adult pro-B acute lymphoblastic leukemia: results of the German multicenter trials GMALL 03/87 and 04/89. Blood. 1998;92:1898-1909. (Pubitemid 28446678)
-
(1998)
Blood
, vol.92
, Issue.6
, pp. 1898-1909
-
-
Ludwig, W.-D.1
Rieder, H.2
Bartram, C.R.3
Heinze, B.4
Schwartz, S.5
Gassmann, W.6
Loffler, H.7
Hossfeld, D.8
Heil, G.9
Handt, S.10
Heyll, A.11
Diedrich, H.12
Fischer, K.13
Weiss, A.14
Volkers, B.15
Aydemir, U.16
Fonatsch, C.17
Gokbuget, N.18
Thiel, E.19
Hoelzer, D.20
more..
-
5
-
-
0141567663
-
Clinico-biologic features and treatment outcome of adult pro-B-ALL patients enrolled in the GIMEMA 0496 study: Absence of the ALL1/AF4 and of the BCR/ABL fusion genes correlates with a significantly better clinical outcome
-
DOI 10.1182/blood-2002-12-3822
-
Cimino G, Elia L, Mancini M, et al. Clinico-biologic features and treatment outcome of adult pro-B-ALL patients enrolled in the GIMEMA 0496 study: absence of the ALL1/AF4 and of the BCR/ABL fusion genes correlates with a significantly better clinical outcome. Blood. 2003;102:2014-2020. (Pubitemid 37122374)
-
(2003)
Blood
, vol.102
, Issue.6
, pp. 2014-2020
-
-
Cimino, G.1
Elia, L.2
Mancini, M.3
Annino, L.4
Anaclerico, B.5
Fazi, P.6
Vitale, A.7
Specchia, G.8
Di Raimondo, F.9
Recchia, A.10
Cuneo, A.11
Mecucci, C.12
Pane, F.13
Saglio, G.14
Foa, R.15
Mandelli, F.16
-
6
-
-
33947375681
-
Age-related differences in leukemia biology and prognosis: The paradigm of MLL-AF4-positive acute lymphoblastic leukemia
-
DOI 10.1038/sj.leu.2404598, PII 2404598
-
Pui CH, Campana D. Age-related differences in leukemia biology and prognosis: the paradigm of MLL-AF4-positive acute lymphoblastic leukemia. Leukemia. 2007;21:593-594. (Pubitemid 46444539)
-
(2007)
Leukemia
, vol.21
, Issue.4
, pp. 593-594
-
-
Pui, C.-H.1
Campana, D.2
-
7
-
-
0029890796
-
Exon/intron structure of the human ALL-1 (MLL) gene involved in translocations to chromosomal region 11q23 and acute leukaemias
-
Nilson I, Lochner K, Siegler G, et al. Exon/intron structure of the human ALL-1 (MLL) gene involved in translocations to chromosomal region 11q23 and acute leukaemias. Br J Haematol. 1996;93:966-972. (Pubitemid 26199341)
-
(1996)
British Journal of Haematology
, vol.93
, Issue.4
, pp. 966-972
-
-
Nilson, I.1
Lochner, K.2
Siegler, G.3
Greil, J.4
Beck, J.D.5
Fey, G.H.6
Marschalek, R.7
-
8
-
-
37049018227
-
Atypical BCR-ABL mRNA transcripts in adult acute lymphoblastic leukemia
-
DOI 10.3324/haematol.11737
-
Burmeister T, Schwartz S, Taubald A, et al. Atypical BCR-ABL mRNA transcripts in adult acute lymphoblastic leukemia. Haematologica. 2007;92:1699-1702. (Pubitemid 350248252)
-
(2007)
Haematologica
, vol.92
, Issue.12
, pp. 1699-1702
-
-
Burmeister, T.1
Schwartz, S.2
Taubald, A.3
Jost, E.4
Lipp, T.5
Schneller, F.6
Diedrich, H.7
Thomssen, H.8
Mey, U.J.M.9
Eucker, J.10
Rieder, H.11
Gokbuget, N.12
Hoelzer, D.13
Thiel, E.14
-
9
-
-
0042527713
-
+ B-cell phenotype
-
DOI 10.1038/sj.leu.2402989
-
Schwartz S, Rieder H, Schläger B, Burmeister T, Fischer L, Thiel E. Expression of the human homologue of rat NG2 in adult acute lymphoblastic leukemia: close association with MLL rearrangement and a CD10(-)/CD24(-)/ CD65s(+)/CD15(+) B-cell phenotype. Leukemia. 2003;17:1589-1595. (Pubitemid 36986961)
-
(2003)
Leukemia
, vol.17
, Issue.8
, pp. 1589-1595
-
-
Schwartz, S.1
Rieder, H.2
Schlager, B.3
Burmeister, T.4
Fischer, L.5
Thiel, E.6
-
10
-
-
0032757710
-
Standardized RT-PCR analysis of fusion gene transcripts from chromosome aberrations in acute leukemia for detection of minimal residual disease. Report of the BIOMED-1 Concerted Action: Investigation of minimal residual disease in acute leukemia
-
van Dongen JJ, Macintyre EA, Gabert JA, et al. Standardized RT-PCR analysis of fusion gene transcripts from chromosome aberrations in acute leukemia for detection of minimal residual disease. Report of the BIOMED-1 Concerted Action: investigation of minimal residual disease in acute leukemia. Leukemia. 1999;13:1901-1928.
-
(1999)
Leukemia
, vol.13
, pp. 1901-1928
-
-
Van Dongen, J.J.1
Macintyre, E.A.2
Gabert, J.A.3
-
11
-
-
33947430631
-
Immunobiological diversity in infant acute lymphoblastic leukemia is related to the occurrence and type of MLL gene rearrangement
-
DOI 10.1038/sj.leu.2404578, PII 2404578
-
Jansen MW, Corral L, van der Velden VH, et al. Immunobiological diversity in infant acute lymphoblastic leukemia is related to the occurrence and type of MLL gene rearrangement. Leukemia. 2007;21:633-641. (Pubitemid 46444545)
-
(2007)
Leukemia
, vol.21
, Issue.4
, pp. 633-641
-
-
Jansen, M.W.J.C.1
Corral, L.2
Van Der Velden, V.H.J.3
Panzer-Grumayer, R.4
Schrappe, M.5
Schrauder, A.6
Marschalek, R.7
Meyer, C.8
Den Boer, M.L.9
Hop, W.J.C.10
Valsecchi, M.G.11
Basso, G.12
Biondi, A.13
Pieters, R.14
Van Dongen, J.J.M.15
-
12
-
-
28844493138
-
- pre-B acute lymphoblastic leukemia (ALL) is a distinct high-risk subgroup of adult ALL associated with a high frequency of MLL aberrations: Results of the German Multicenter Trials for Adult ALL (GMALL)
-
DOI 10.1182/blood-2005-05-1866
-
Gleissner B, Gökbuget N, Rieder H, et al. CD10-pre-B acute lymphoblastic leukemia (ALL) is a distinct high-risk subgroup of adult ALL associated with a high frequency of MLL aberrations: results of the German Multicenter Trials for Adult ALL (GMALL). Blood. 2005;106:4054-4056. (Pubitemid 41775909)
-
(2005)
Blood
, vol.106
, Issue.13
, pp. 4054-4056
-
-
Gleissner, B.1
Goekbuget, N.2
Rieder, H.3
Arnold, R.4
Schwartz, S.5
Diedrich, H.6
Schoch, C.7
Heinze, B.8
Fonatsch, C.9
Bartram, C.R.10
Hoelzer, D.11
Thiel, E.12
-
13
-
-
19944427410
-
Diagnostic tool for the identification of MLL rearrangements including unknown partner genes
-
DOI 10.1073/pnas.0406994102
-
Meyer C, Schneider B, Reichel M, et al. Diagnostic tool for the identification of MLL rearrangements including unknown partner genes. Proc Natl Acad Sci U S A. 2005;102:449-454. (Pubitemid 40116787)
-
(2005)
Proceedings of the National Academy of Sciences of the United States of America
, vol.102
, Issue.2
, pp. 449-454
-
-
Meyer, C.1
Schneider, B.2
Reichel, M.3
Angermueller, S.4
Strehl, S.5
Schnittger, S.6
Schoch, C.7
Jansen, M.W.J.C.8
Van Dongen, J.J.9
Pieters, R.10
Haas, O.A.11
Dingermann, T.12
Klingebiel, T.13
Marschalek, R.14
-
14
-
-
33847234331
-
Spliced MLL fusions: A novel mechanism to generate functional chimeric MLL-MLLT1 transcripts in t(11;19)(q23;p13.3) leukemia [16]
-
DOI 10.1038/sj.leu.2404542, PII 2404542
-
Meyer C, Burmeister T, Strehl S, et al. Spliced MLL fusions: a novel mechanism to generate functional chimeric MLL-MLLT1 transcripts in t(11;19)(q23;p13.3) leukemia. Leukemia. 2007;21:588-590. (Pubitemid 46306583)
-
(2007)
Leukemia
, vol.21
, Issue.3
, pp. 588-590
-
-
Meyer, C.1
Burmeister, T.2
Strehl, S.3
Schneider, B.4
Hubert, D.5
Zach, O.6
Haas, O.7
Klingebiel, T.8
Dingermann, T.9
Marschalek, R.10
-
15
-
-
0033565246
-
Rapid isolation of chromosomal breakpoints from patients with t(4;11) acute lymphoblastic leukemia: Implications for basic and clinical research
-
Reichel M, Gillert E, Breitenlohner I, et al. Rapid isolation of chromosomal breakpoints from patients with t(4;11) acute lymphoblastic leukemia: implications for basic and clinical research. Cancer Res. 1999;59:3357-3362. (Pubitemid 29334491)
-
(1999)
Cancer Research
, vol.59
, Issue.14
, pp. 3357-3362
-
-
Reichel, M.1
Gillert, E.2
Breitenlohner, I.3
Repp, R.4
Greil, J.5
Beck, J.D.6
Fey, G.H.7
Marschalek, R.8
-
16
-
-
0032894303
-
Childhood acute lymphoblastic leukemia with the MLL-ENL fusion and t(11;19)(q23;p13.3) Translocation
-
Rubnitz JE, Camitta BM, Mahmoud H, et al. Childhood acute lymphoblastic leukemia with the MLL-ENL fusion and t(11;19)(q23;p13.3) translocation. J Clin Oncol. 1999;17:191-196. (Pubitemid 29022393)
-
(1999)
Journal of Clinical Oncology
, vol.17
, Issue.1
, pp. 191-196
-
-
Rubnitz, J.E.1
Camitta, B.M.2
Mahmoud, H.3
Raimondi, S.C.4
Carroll, A.J.5
Borowitz, M.J.6
Shuster, J.J.7
Link, M.P.8
Pullen, D.J.9
Downing, J.R.10
Behm, F.G.11
Pui, C.-H.12
-
17
-
-
0037099537
-
LCX, leukemia-associated protein with a CXXC domain, is fused to MLL in acute myeloid leukemia with trilineage dysplasia having t(10;11)(q22;q23)
-
Ono R, Taki T, Taketani T, Taniwaki M, Kobayashi H, Hayashi Y. LCX, leukemia-associated protein with a CXXC domain, is fused to MLL in acute myeloid leukemia with trilineage dysplasia having t(10;11)(q22;q23). Cancer Res. 2002;62:4075-4080. (Pubitemid 34791077)
-
(2002)
Cancer Research
, vol.62
, Issue.14
, pp. 4075-4080
-
-
Ono, R.1
Taki, T.2
Taketani, T.3
Taniwaki, M.4
Kobayashi, H.5
Hayashi, Y.6
-
18
-
-
0037350661
-
TET1, a member of a novel protein family, is fused to MLL in acute myeloid leukemia containing the t(10;11)(q22;23) [3]
-
DOI 10.1038/sj.leu.2402834
-
Lorsbach RB, Moore J, Mathew S, Raimondi SC, Mukatira ST, Downing JR. TET1, a member of a novel protein family, is fused to MLL in acute myeloid leukemia containing the t(10;11)(q22;q23). Leukemia. 2003;17:637-641. (Pubitemid 36395661)
-
(2003)
Leukemia
, vol.17
, Issue.3
, pp. 637-641
-
-
Lorsback, R.B.1
Moore, J.2
Mathew, S.3
Raimondi, S.C.4
Mukatira, S.T.5
Downing, J.R.6
-
19
-
-
31444447072
-
Characterization of fusion partner genes in 114 patients with de novo acute myeloid leukemia and MLL rearrangement
-
Shih LY, Liang DC, Fu JF, et al. Characterization of fusion partner genes in 114 patients with de novo acute myeloid leukemia and MLL rearrangement. Leukemia. 2006;20:218-223.
-
(2006)
Leukemia
, vol.20
, pp. 218-223
-
-
Shih, L.Y.1
Liang, D.C.2
Fu, J.F.3
-
20
-
-
0034051681
-
Mutations in ACTN4, encoding α-actinin-4, cause familial focal segmental glomerulosclerosis
-
Kaplan JM, Kim SH, North KN, et al. Mutations in ACTN4, encoding α-actinin-4, cause familial focal segmental glomerulosclerosis. Nat Genet. 2000;24:251-256.
-
(2000)
Nat Genet
, vol.24
, pp. 251-256
-
-
Kaplan, J.M.1
Kim, S.H.2
North, K.N.3
-
21
-
-
0035872398
-
A point mutation in the α-actinin-4 gene generates an antigenic peptide recognized by autologous cytolytic T lymphocytes on a human lung carcinoma
-
Echchakir H, Mami-Chouaib F, Vergnon I, et al. A point mutation in the α-actinin-4 gene generates an antigenic peptide recognized by autologous cytolytic T lymphocytes on a human lung carcinoma. Cancer Res. 2001;61:4078-4083. (Pubitemid 32720974)
-
(2001)
Cancer Research
, vol.61
, Issue.10
, pp. 4078-4083
-
-
Echchakir, H.1
Mami-Chouaib, F.2
Vergnon, I.3
Baurain, J.-F.4
Karanikas, V.5
Chouaib, S.6
Coulie, P.G.7
-
22
-
-
0141816710
-
AML with 11q23/MLL abnormalities as defined by the WHO classification: Incidence, partner chromosomes, FAB subtype, age distribution, and prognostic impact in an unselected series of 1897 cytogenetically analyzed AML cases
-
Schoch C, Schnittger S, Klaus M, Kern W, Hiddemann W, Haferlach T. AML with 11q23/MLL abnormalities as defined by the WHO classification: incidence, partner chromosomes, FAB subtype, age distribution, and prognostic impact in an unselected series of 1897 cytogenetically analyzed AML cases. Blood. 2003;102:2395-2402.
-
(2003)
Blood
, vol.102
, pp. 2395-2402
-
-
Schoch, C.1
Schnittger, S.2
Klaus, M.3
Kern, W.4
Hiddemann, W.5
Haferlach, T.6
-
23
-
-
1342305488
-
Malignant hematopoietic cell lines: In vitro models for the study of MLL gene alterations
-
DOI 10.1038/sj.leu.2403236
-
Drexler HG, Quentmeier H, MacLeod RA. Malignant hematopoietic cell lines: in vitro models for the study of MLL gene alterations. Leukemia. 2004;18:227-232. (Pubitemid 38247741)
-
(2004)
Leukemia
, vol.18
, Issue.2
, pp. 227-232
-
-
Drexler, H.G.1
Quentmeier, H.2
MacLeod, R.A.F.3
|