Severity of atypical absence phenotype in GABAB transgenic mice is subunit specific

Epilepsy and Behavior

Volumn 14, Issue 4, 2009, Pages 577-581

  Stewart, Lee S ^a   Wu, Ying ^a   Eubanks, James H ^c   Han, Hua ^a   Leschenko, Yevgen ^a   Perez Velazquez, Jose L ^a,b   Cortez, Miguel A ^a,b   Snead III, O Carter ^a,b  

^a HOSPITAL FOR SICK CHILDREN   (Canada)

^b UNIVERSITY OF TORONTO   (Canada)

^c UNIVERSITY HEALTH NETWORK   (Canada)

Author keywords

Atypical absence seizures; Behavior; GABABR1b receptors; Long term potentiation; Slow spike and wave discharges

Indexed keywords

4 AMINOBUTYRIC ACID B RECEPTOR; RECEPTOR SUBUNIT;

ABSENCE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTORADIOGRAPHY; COGNITIVE DEFECT; CONTROLLED STUDY; DISEASE SEVERITY; ELECTROGRAPHY; EPILEPTIC DISCHARGE; FOREBRAIN; HIPPOCAMPUS; HYPOTHESIS; IMMUNOBLOTTING; INHIBITORY POSTSYNAPTIC POTENTIAL; LONG TERM POTENTIATION; MOUSE; NERVE CELL PLASTICITY; NONHUMAN; PHENOTYPE; PROTEIN EXPRESSION; SPIKE WAVE; TRANSGENIC MOUSE;

6-CYANO-7-NITROQUINOXALINE-2,3-DIONE; ANIMALS; ANIMALS, NEWBORN; AUTORADIOGRAPHY; COGNITION DISORDERS; DISEASE MODELS, ANIMAL; ELECTRIC STIMULATION; ELECTROENCEPHALOGRAPHY; EPILEPSY, ABSENCE; EXCITATORY AMINO ACID ANTAGONISTS; HIPPOCAMPUS; INHIBITORY POSTSYNAPTIC POTENTIALS; LONG-TERM POTENTIATION; MAZE LEARNING; MICE; MICE, TRANSGENIC; NEURONS; ORGANOPHOSPHORUS COMPOUNDS; PATCH-CLAMP TECHNIQUES; PHENOTYPE; PROTEIN BINDING; RECEPTORS, GABA-B; TRITIUM; VALINE;

EID: 63749110733     PISSN: 15255050     EISSN: 15255069     Source Type: Journal    
DOI: 10.1016/j.yebeh.2009.01.019     Document Type: Article

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