The transcription factor Eya2 prevents pressure overload-induced adverse cardiac remodeling

Journal of Molecular and Cellular Cardiology

Volumn 46, Issue 4, 2009, Pages 596-605

  Lee, Seung Hee ^a   Yang, Dong Kwon ^a   Choi, Bo Youn ^a   Lee, Young Hoon ^b   Kim, Seon Young ^c   Jeong, Dongtak ^d   Hajjar, Roger J ^d   Park, Woo Jin ^a  

^a Gwangju Institute of Science and Technology (GIST)   (South Korea)

^b Chonbuk National University   (South Korea)

^c Korea Research Institute of Bioscience and Biotechnology (KRIBB)   (South Korea)

^d NEW YORK UNIVERSITY   (United States)

Author keywords

Akt; Cardiac hypertrophy; Eyes absent 2; Heart failure

Indexed keywords

MAMMALIAN TARGET OF RAPAMYCIN; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR EYA2; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BIOGENESIS; CELL SURFACE; CONTROLLED STUDY; GENE EXPRESSION PROFILING; HEART FAILURE; HEART FUNCTION; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE OVERLOAD; HEART VENTRICLE PRESSURE; HEART VENTRICLE REMODELING; HEART WEIGHT; MALE; MITOCHONDRIAL RESPIRATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; SIGNAL TRANSDUCTION;

ANIMALS; CARDIOMEGALY; FIBROSIS; GENE EXPRESSION PROFILING; GENE EXPRESSION REGULATION; HEART FAILURE; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MICE; MICE, TRANSGENIC; NUCLEAR PROTEINS; ORGAN SPECIFICITY; PHOSPHATIDYLINOSITOL 3-KINASES; PRESSURE; PROTEIN KINASES; PROTEIN TYROSINE PHOSPHATASES; PROTO-ONCOGENE PROTEINS C-AKT; SIGNAL TRANSDUCTION; STRESS, PHYSIOLOGICAL; TIME FACTORS; TOR SERINE-THREONINE KINASES; TRANSCRIPTION FACTORS; VENTRICULAR REMODELING;

MUS MUSCULUS;

EID: 61449150343     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2008.12.021     Document Type: Article

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