Glomerular endothelial NOS (eNOS) expression in type 2 diabetic patients with nephropathy

Nephrology Dialysis Transplantation

Volumn 23, Issue 9, 2008, Pages 3037-

  Komers, Radko ^a   Anderson, Sharon ^a  

^a OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ENDOTHELIAL NITRIC OXIDE SYNTHASE; NITRIC OXIDE;

DIABETIC NEPHROPATHY; ENDOTHELIAL DYSFUNCTION; GLOMERULUS; LETTER; NON INSULIN DEPENDENT DIABETES MELLITUS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEINURIA;

BIOLOGICAL AVAILABILITY; DIABETES MELLITUS, TYPE 2; DIABETIC NEPHROPATHIES; HUMANS; IMMUNOHISTOCHEMISTRY; KIDNEY GLOMERULUS; NITRIC OXIDE SYNTHASE TYPE III; PHOSPHORYLATION;

EID: 58549087946     PISSN: 09310509     EISSN: 14602385     Source Type: Journal    
DOI: 10.1093/ndt/gfn174     Document Type: Letter

References (5)

1. Komers R, Anderson S. The paradoxes of nitric oxide in the diabetic kidney. Am J Physiol 2003; 284: F1121-F1137
2. Hohenstein B, Hugo CP, Hausknecht B et al. Analysis of NO-synthase expression and clinical risk factors in human diabetic nephropathy. Nephrol Dial Transplant 2008; 4: 1346-1354
3. Govers R, Rabelink TJ. Cellular regulation of endothelial nitric oxide synthase. Am J Physiol Renal Physiol 2001; 280: F193-F206
4. Komers R, Schutzer WE, Reed JF et al. Altered endothelial nitric oxide synthase targeting and conformation and caveolin-1 expression in the diabetic kidney. Diabetes 2006; 55: 1651-1659
5. Satoh M, Fujimoto S, Haruna Y et al. NAD(P)H oxidase and uncoupled nitric oxide synthase are major sources of glomerular superoxide in rats with experimental diabetic nephropathy. Am J Physiol Renal Physiol 2005; 288: F1144-F1152