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Volumn 1150, Issue , 2008, Pages 187-189

IFN-α can both protect against and promote the development of type 1 diabetes

Author keywords

IFN ; Poly:IC; Type 1 diabetes

Indexed keywords

ALPHA INTERFERON; GLUCOSE; POLYINOSINIC POLYCYTIDYLIC ACID;

EID: 57749193697     PISSN: 00778923     EISSN: 17496632     Source Type: Book Series    
DOI: 10.1196/annals.1447.031     Document Type: Conference Paper
Times cited : (15)

References (7)
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    • Wen, L., J. Peng, Z. Li & F.S. Wong. 2004. The effect of innate immunity on autoimmune diabetes and the expression of Toll-like receptors on pancreatic islets. J. Immunol. 172 : 3173 3180.
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    • Wen, L.1    Peng, J.2    Li, Z.3    Wong, F.S.4
  • 4
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    • Islet expression of interferon-alpha precedes diabetes in both the BB rat and streptozotocin-treated mice
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    • Huang, X., B. Hultgren, N. Dybdal & T.A. Stewart. 1994. Islet expression of interferon-alpha precedes diabetes in both the BB rat and streptozotocin-treated mice. Immunity 1 : 469 478.
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    • TLR activation synergizes with Kilham rat virus infection to induce diabetes in BBDR rats
    • Zipris, D. et al. 2005. TLR activation synergizes with Kilham rat virus infection to induce diabetes in BBDR rats. J. Immunol. 174 : 131 142.
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    • Induction of insulitis by glutamic acid decarboxylase peptide-specific and HLA-DQ8-restricted CD4(+) T cells from human DQ transgenic mice
    • Wen, L. et al. 1998. Induction of insulitis by glutamic acid decarboxylase peptide-specific and HLA-DQ8-restricted CD4(+) T cells from human DQ transgenic mice. J. Clin. Invest. 102 : 947 957.
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    • Wen, L.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.