Endotoxin induces a delayed loss of TH-IR neurons in substantia nigra and motor behavioral deficits

NeuroToxicology

Volumn 29, Issue 5, 2008, Pages 864-870

  Liu, Yuxin ^a,b   Qin, Liya ^b   Wilson, Belinda ^a   Wu, Xuefei ^a   Qian, Li ^a,c   Granholm, Ann Charlotte ^d   Crews, Fulton T ^b   Hong, Jau Shyong ^a  

^a NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

^b UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF NORTH CAROLINA   (United States)

^d MEDICAL UNIVERSITY OF SOUTH CAROLINA   (United States)

Author keywords

Lipopolysaccharide; Neurodegeneration; Parkinson's disease; Rotarod test; TH IR neurons

Indexed keywords

ALPHA SYNUCLEIN; CARBIDOPA; ENDOTOXIN; LEVODOPA; LIPOPOLYSACCHARIDE; TYROSINE 3 MONOOXYGENASE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BEHAVIOR CHANGE; BRAIN REGION; CELL LOSS; CONTROLLED STUDY; DOPAMINERGIC NERVE CELL; DRUG EFFECT; FEMALE; IMMUNOHISTOCHEMISTRY; MALE; MOTOR DYSFUNCTION; MOTOR PERFORMANCE; MOUSE; NEUROTOXICITY; NONHUMAN; OPEN FIELD TEST; PRIORITY JOURNAL; ROTAROD TEST; SEX DIFFERENCE; SUBSTANTIA NIGRA;

ANALYSIS OF VARIANCE; ANIMALS; DOPAMINE AGENTS; ENDOTOXINS; EXPLORATORY BEHAVIOR; FEMALE; LEVODOPA; LIPOPOLYSACCHARIDES; MALE; MICE; MICE, INBRED C57BL; MOTOR ACTIVITY; NEURONS; ROTAROD PERFORMANCE TEST; SEX FACTORS; SUBSTANTIA NIGRA; TIME FACTORS; TYROSINE 3-MONOOXYGENASE;

MUS;

EID: 52149123921     PISSN: 0161813X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuro.2008.02.014     Document Type: Article

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