Transient activation of P38 MAP kinase and up-regulation of Pim-1 kinase in cardiac hypertrophy despite no activation of AMPK

Journal of Molecular and Cellular Cardiology

Volumn 45, Issue 3, 2008, Pages 404-410

  Lei, Biao ^a   Chess, David J ^a   Keung, Wendy ^b   O'Shea, Karen M ^a   Lopaschuk, Gary D ^b   Stanley, William C ^a  

^a UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF ALBERTA   (Canada)

Author keywords

Akt; AMP activated protein kinase; Heart failure; Hypertrophy; P38 MAPK; Pim 1

Indexed keywords

HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; MITOGEN ACTIVATED PROTEIN KINASE P38; PROTEIN KINASE; PROTEIN KINASE B; PROTEIN KINASE PIM 1; THREONINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME PHOSPHORYLATION; ENZYME SUBUNIT; HEART DILATATION; HEART LEFT VENTRICLE CONTRACTILITY; HEART LEFT VENTRICLE HYPERTROPHY; HEART MUSCLE METABOLISM; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PROTEIN INTERACTION; UPREGULATION;

AMINO ACID SEQUENCE; AMP-ACTIVATED PROTEIN KINASES; ANIMALS; DISEASE MODELS, ANIMAL; ENZYME ACTIVATION; HYPERTROPHY, LEFT VENTRICULAR; MALE; MICE; MICE, INBRED C57BL; MOLECULAR SEQUENCE DATA; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PROTO-ONCOGENE PROTEINS C-PIM-1; TIME FACTORS; UP-REGULATION;

MUS;

EID: 50849099136     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2008.06.008     Document Type: Article

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