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Volumn 88, Issue 22, 2008, Pages 1540-1542

Expression and significance of glypican-3 in colorectal cancer

Author keywords

Glypican 3; Immunohistochemistry; Intestinal neoplasms

Indexed keywords

GLYPICAN 3;

EID: 50249154009     PISSN: 03762491     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (4)

References (13)
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    • Pilia G, Hughes-Benzie RM, MacKenzie A, et al. Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome. Nature Genet, 1996,12 f31:241-247.
  • 2
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  • 3
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  • 4
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    • Chinese source.
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    • Chinese source.
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  • 6
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    • The loss of Glypican-3 induces alterations in Wnt signaling
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  • 7
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    • Cellular changes resulting from forced expression of glypican23 in hepatocellular carcinoma cells. Histol Histopathol
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  • 8
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    • Glypican-3 promotes the growth of hepatocellular carcinoma by stimulating canonical Wnt signaling
    • Capurro MI, Xiang YY, Lobe C, et al. Glypican-3 promotes the growth of hepatocellular carcinoma by stimulating canonical Wnt signaling. Cancer Res, 2005, 65:6245-6254.
    • (2005) Cancer Res , vol.65 , pp. 6245-6254
    • Capurro, M.I.1    Xiang, Y.Y.2    Lobe, C.3
  • 9
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    • Frequent silencing of the GLYPICAN-3 gene in ovarian cancer cell lines
    • Lin H, Huber R, Schlessinger D, et al. Frequent silencing of the GLYPICAN-3 gene in ovarian cancer cell lines. Cancer Res, 1999, 59: 807-810.
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  • 10
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    • oci glypican-3, a glypican encoded by a gene that is mutated in the Simpson- Golabi-Behmel overgrowth syndrome, induces apoptosis in a cell line-specific manner
    • Gonzalez AD, Kaya M, Shi W, et al. oci glypican-3, a glypican encoded by a gene that is mutated in the Simpson- Golabi-Behmel overgrowth syndrome, induces apoptosis in a cell line-specific manner. J Cell Biol, 1998, 141:1407-1414.
    • (1998) J Cell Biol , vol.141 , pp. 1407-1414
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  • 11
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    • Peters, M.G.1    Farias, E.2    Colombo, L.3
  • 12
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    • Blocking endogenous glypican-3 expression releases Hep 3B cells from G1 arrest
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    • Capurro, M.1    Wanless, I.R.2    Sherman, M.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.