Endothelin-1-mediated coronary vasoconstriction deteriorates myocardial depression in hearts isolated from lipopolysaccharide-treated rats: Interaction with nitric oxide

Clinical and Experimental Pharmacology and Physiology

Volumn 31, Issue 9, 2004, Pages 571-574

  Tu, Jie ^a   Shan, Qixian ^a   Jin, Hongfeng ^a   Bourreau, Jean Pierre ^b   Xia, Qiang ^a  

^a ZHEJIANG UNIVERSITY SCHOOL OF MEDICINE   (China)

^b UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

Coronary perfusion pressure; Endothelin; Lipopolysaccharide; Myocardial depression; Nitric oxide

Indexed keywords

AMINOGUANIDINE; CYCLO(DEXTRO TRYPTOPHYL DEXTRO ASPARTYLPROLYL DEXTRO VALYLLEUCYL); ENDOTHELIN 1; LIPOPOLYSACCHARIDE; NITRIC OXIDE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONCENTRATION RESPONSE; CONTROLLED STUDY; CORONARY ARTERY BLOOD FLOW; CORONARY ARTERY CONSTRICTION; HEART LEFT VENTRICLE PRESSURE; HEART MUSCLE CONTRACTILE FORCE; HEART RATE; MALE; MYOCARDIAL DISEASE; NONHUMAN; PROTEIN PROTEIN INTERACTION; RADIOIMMUNOASSAY; RAT;

ANIMALS; CORONARY VESSELS; ENDOTHELIN-1; GUANIDINES; LIPOPOLYSACCHARIDES; MALE; MYOCARDIAL CONTRACTION; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; PEPTIDES, CYCLIC; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, ENDOTHELIN A; TIME FACTORS; VASOCONSTRICTION;

EID: 4944232609     PISSN: 03051870     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1440-1681.2004.04049.x     Document Type: Article

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