Physiological and pathological actions of Calpains in glutamatergic neurons

Science Signaling

Volumn 1, Issue 23, 2008, Pages

  Liu, Jing ^a   Liu, Ming Cheng ^a,b   Wang, Kevin K W ^a,b  

^a UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

^b BANYAN BIOMARKERS INC   (United States)

Author keywords

Action potential; Biomarkers; Calcium; Calpain; Glutamate; Movie; Neurotoxicity; Signal transduction; Synaptic plasticity

Indexed keywords

CALPAIN; GLUTAMIC ACID;

ANIMAL; ARTICLE; CELL DEATH; HUMAN; NERVE CELL; NERVE CELL PLASTICITY; NERVE ENDING; PATHOLOGY; PHYSIOLOGY; SYNAPSE; SYNAPTIC TRANSMISSION;

ANIMALS; CALPAIN; CELL DEATH; GLUTAMIC ACID; HUMANS; NERVE ENDINGS; NEURONAL PLASTICITY; NEURONS; SYNAPSES; SYNAPTIC TRANSMISSION;

EID: 48649101596     PISSN: 19450877     EISSN: 19379145     Source Type: Journal    
DOI: 10.1126/scisignal.123tr3     Document Type: Review

References (4)

1. Citation: J. Liu, M. C. Liu, K. K. W. Wang, Physiological and Pathological Actions of Calpains in Glutamatergic Neurons. Sci. Signal. 1, tr3 (2008).
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