Isoniazid-induced reduction in GABAergic neurotransmission alters the function of the cerebellar cortical circuit

Neuroscience

Volumn 154, Issue 2, 2008, Pages 710-719

  Carta, M ^a   Murru, L ^a   Barabino, E ^a   Talani, G ^a   Sanna, E ^a   Biggio, G ^a,b  

^a UNIVERSITY OF CAGLIARI   (Italy)

^b CNR   (Italy)

Author keywords

cerebellum; excitability; GABA; glutamic acid decarboxylase; isoniazid; synapse

Indexed keywords

4 AMINOBUTYRIC ACID; GLUTAMATE DECARBOXYLASE; ISONIAZID;

AMINO ACID SYNTHESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN CELL; BRAIN FUNCTION; BRAIN SLICE; CEREBELLUM CORTEX; CONTROLLED STUDY; ENZYME INHIBITION; GABAERGIC SYSTEM; GABAERGIC TRANSMISSION; GRANULE CELL; INFORMATION PROCESSING; MALE; NERVE CELL EXCITABILITY; NONHUMAN; PATCH CLAMP; PRIORITY JOURNAL; PURKINJE CELL; RAT; VOLTAGE CLAMP;

ANIMALS; ANTITUBERCULAR AGENTS; CEREBELLAR CORTEX; ELECTROPHYSIOLOGY; ENZYME INHIBITORS; EXCITATORY POSTSYNAPTIC POTENTIALS; GAMMA-AMINOBUTYRIC ACID; GLUTAMATE DECARBOXYLASE; ISONIAZID; MALE; NERVE NET; PURKINJE CELLS; RATS; RATS, SPRAGUE-DAWLEY; SYNAPSES; SYNAPTIC TRANSMISSION;

EID: 46549086792     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2008.02.024     Document Type: Article

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