Hydroquinone, a major component in cigarette smoke, reduces IFN-γ production in antigen-primed lymphocytes

Archives of Pharmacal Research

Volumn 31, Issue 3, 2008, Pages 337-341

  Choi, Jin Myung ^a   Cho, Young Chang ^a   Cho, Won Jea ^a   Kim, Tae Sung ^b   Kang, Bok Yun ^a  

^a Chonnam National University   (South Korea)

^b Korea University   (South Korea)

Author keywords

AP 1; Hydroquinone; IFN ; NF B; T cell

Indexed keywords

GAMMA INTERFERON; HEMOCYANIN; HYDROQUINONE; HYDROQUINONE DERIVATIVE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; KEYHOLE LIMPET HEMOCYANIN; KEYHOLE-LIMPET HEMOCYANIN; MESSENGER RNA; TRANSCRIPTION FACTOR AP 1;

ANIMAL; ARTICLE; C57BL MOUSE; CD4+ T LYMPHOCYTE; CELL CULTURE; DOSE RESPONSE; DRUG EFFECT; FEMALE; GENETIC TRANSCRIPTION; GENETIC TRANSFECTION; GENETICS; IMMUNOLOGY; LYMPHOCYTE; METABOLISM; MOUSE; PROMOTER REGION; SMOKE; TH1 CELL; TOBACCO;

ANIMALS; CD4-POSITIVE T-LYMPHOCYTES; CELLS, CULTURED; DOSE-RESPONSE RELATIONSHIP, DRUG; FEMALE; HEMOCYANIN; HYDROQUINONES; INTERFERON TYPE II; LYMPHOCYTES; MICE; MICE, INBRED C57BL; NF-KAPPA B; PROMOTER REGIONS (GENETICS); RNA, MESSENGER; SMOKE; TH1 CELLS; TOBACCO; TRANSCRIPTION FACTOR AP-1; TRANSCRIPTION, GENETIC; TRANSFECTION;

EID: 46249104981     PISSN: 02536269     EISSN: 19763786     Source Type: Journal    
DOI: 10.1007/s12272-001-1161-1     Document Type: Article

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