Methionine synthase reductase deficiency results in adverse reproductive outcomes and congenital heart defects in mice

Molecular Genetics and Metabolism

Volumn 94, Issue 3, 2008, Pages 336-342

  Deng, Liyuan ^a   Elmore, C Lee ^b   Lawrance, Andrea K ^a   Matthews, Rowena G ^b   Rozen, Rima ^a  

^a RESEARCH INSTITUTE OF THE MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

^b UNIVERSITY OF MICHIGAN   (United States)

Author keywords

Congenital heart defects; Folate; Methionine; Pregnancy complications; Reproductive outcome

Indexed keywords

METHIONINE SYNTHASE REDUCTASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONGENITAL HEART MALFORMATION; EMBRYO; EMBRYO DEVELOPMENT; ENZYME DEFICIENCY; FEMALE; GENETIC POLYMORPHISM; GENOTYPE; HEART DEVELOPMENT; HEART VENTRICLE SEPTUM DEFECT; MALE; MOUSE; NONHUMAN; OUTCOMES RESEARCH; PREGNANCY COMPLICATION; PRIORITY JOURNAL; REPRODUCTION; SEX DIFFERENCE;

ANIMALS; EMBRYO, MAMMALIAN; FEMALE; FERREDOXIN-NADP REDUCTASE; GENOTYPE; HEART DEFECTS, CONGENITAL; INCIDENCE; MALE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; PREGNANCY; PREGNANCY OUTCOME; REPRODUCTION; SEX CHARACTERISTICS;

MUS;

EID: 44949122500     PISSN: 10967192     EISSN: 10967206     Source Type: Journal    
DOI: 10.1016/j.ymgme.2008.03.004     Document Type: Article

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