Effects of MEK5/ERK5 association on small ubiquitin-related modification of ERK5: Implications for diabetic ventricular dysfunction after myocardial infarction

Circulation Research

Volumn 102, Issue 11, 2008, Pages 1416-1425

  Shishido, Tetsuro ^a   Woo, Chang Hoon ^a   Ding, Bo ^a   McClain, Carolyn ^a   Molina, Carlos A ^c   Yan, Chen ^a   Yang, Jay ^b   Abe, Jun Ichi ^a  

^a UNIVERSITY OF ROCHESTER SCHOOL OF MEDICINE AND DENTISTRY   (United States)

^b Columbia University P and S ^*  (United States)

^c MONTCLAIR STATE UNIVERSITY   (United States)

Author keywords

Apoptosis; Diabetes; ERK5; Myocardial infarction; SUMOylation

Indexed keywords

GLUCOSE; HYDROGEN PEROXIDE; MITOGEN ACTIVATED PROTEIN KINASE 7; MITOGEN ACTIVATED PROTEIN KINASE KINASE 5; PROTEIN INHIBITOR OF ACTIVATED STAT; PROTEIN UBC9; SMALL INTERFERING RNA; OXIDIZING AGENT; STREPTOZOCIN; SUMO PROTEIN; UBIQUITIN CONJUGATING ENZYME; UBIQUITIN CONJUGATING ENZYME UBC9; UBIQUITIN-CONJUGATING ENZYME UBC9;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DEATH; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ACTIVITY; GENETIC TRANSCRIPTION; HEART FUNCTION; HEART INFARCTION; HEART LEFT VENTRICLE FAILURE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN INTERACTION; STREPTOZOCIN DIABETES; SUMOYLATION; ANIMAL; APOPTOSIS; CELL CULTURE; CHEMICALLY INDUCED DISORDER; DISEASE MODEL; DRUG EFFECT; EXPERIMENTAL DIABETES MELLITUS; GENETICS; HEART MUSCLE CELL; HEART VENTRICLE FUNCTION; HYPERGLYCEMIA; METABOLISM; PATHOPHYSIOLOGY;

ANIMALS; APOPTOSIS; CELLS, CULTURED; DIABETES MELLITUS, EXPERIMENTAL; DISEASE MODELS, ANIMAL; ENZYME ACTIVATION; HYDROGEN PEROXIDE; HYPERGLYCEMIA; MAP KINASE KINASE 5; MICE; MITOGEN-ACTIVATED PROTEIN KINASE 7; MYOCARDIAL INFARCTION; MYOCYTES, CARDIAC; OXIDANTS; SMALL UBIQUITIN-RELATED MODIFIER PROTEINS; STREPTOZOCIN; TRANSCRIPTION, GENETIC; UBIQUITIN-CONJUGATING ENZYMES; VENTRICULAR DYSFUNCTION;

EID: 44949114361     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.107.168138     Document Type: Article

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