How are podocytes affected in nail-patella syndrome?

Pediatric Nephrology

Volumn 23, Issue 7, 2008, Pages 1017-1020

  Witzgall, Ralph ^a  

^a UNIVERSITY OF REGENSBURG   (Germany)

Author keywords

LMX1B; Mouse model; Podocyte foot process; Promotor analysis; Slit diaphragm; Targetgenes

Indexed keywords

COLLAGEN TYPE 4; COLLAGEN TYPE 4 ALPHA3 CHAIN; COLLAGEN TYPE 4 ALPHA4 CHAIN; NEPHRIN; PODOCIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR LMX1B; VASCULOTROPIN;

CELL DAMAGE; CELL DIFFERENTIATION; GENE FUNCTION; GENE INACTIVATION; GENE MUTATION; GENETIC DISORDER; GLOMERULUS BASEMENT MEMBRANE; HUMAN; KIDNEY DISEASE; KNOCKOUT MOUSE; NAIL PATELLA SYNDROME; NONHUMAN; NOTE; PODOCYTE; PRIORITY JOURNAL; PROGNOSIS; PROTEIN LOCALIZATION; RISK FACTOR; SYMPTOMATOLOGY; UPREGULATION;

ANIMALS; CELL DIFFERENTIATION; DISEASE MODELS, ANIMAL; HOMEODOMAIN PROTEINS; HUMANS; KIDNEY DISEASES; MICE; MICE, KNOCKOUT; MUTATION; NAIL-PATELLA SYNDROME; PODOCYTES; TRANSCRIPTION FACTORS;

EID: 44449146510     PISSN: 0931041X     EISSN: None     Source Type: Journal    
DOI: 10.1007/s00467-007-0714-9     Document Type: Note

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