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Journal of Physiology

Volumn 586, Issue 9, 2008, Pages 2249-2250

Persistent pain: The contribution of NaV1.9

(2) Smith, Ewan St John a Momin, Aliakmal b

a MAX DELBRÜCK CENTER FOR MOLECULAR MEDICINE (Germany)

b UNIVERSITY OF CAMBRIDGE (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN KINASE C; SODIUM CHANNEL BLOCKING AGENT; SODIUM ION; TETRODOTOXIN; VOLTAGE GATED SODIUM CHANNEL; GUANOSINE 5' O (3 THIOTRIPHOSPHATE); SODIUM CHANNEL NAV1.8; SODIUM CHANNEL NAV1.9;

ARTICLE; ENZYME ACTIVATION; GENE DISRUPTION; HETEROLOGOUS EXPRESSION; HYPERALGESIA; INFLAMMATION; NERVE CELL MEMBRANE POTENTIAL; NERVE CELL MEMBRANE STEADY POTENTIAL; NERVE CELL STIMULATION; NERVE EXCITABILITY; NONHUMAN; PAIN; PRIORITY JOURNAL; SODIUM CURRENT; UPREGULATION; CELL MEMBRANE POTENTIAL; DRUG TARGETING; HUMAN; INFLAMMATORY PAIN; NEUROPATHOLOGY; NOCICEPTION; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; VOLTAGE CLAMP TECHNIQUE;

ANIMALS; HUMANS; ION CHANNEL GATING; MODELS, NEUROLOGICAL; NEURONS, AFFERENT; NEUROPEPTIDES; NOCICEPTORS; PAIN; SODIUM CHANNELS;

EID: 42949160506 PISSN: 00223751 EISSN: 14697793 Source Type: Journal
DOI: 10.1113/jphysiol.2008.152520 Document Type: Article

Times cited : (4)

References (7)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.