Increase in intracellular free/bound NAD[P]H as a cause of Cd-induced oxidative stress in the HepG2 cells

Toxicology

Volumn 247, Issue 1, 2008, Pages 6-10

  Yang, M S ^a   Li, D ^b   Lin, T ^a   Zheng, J J ^b   Zheng, W ^b   Qu, J Y ^b  

^a HONG KONG BAPTIST UNIVERSITY   (Hong Kong)

^b HONG KONG UNIVERSITY OF SCIENCE AND TECHNOLOGY   (Hong Kong)

Author keywords

Cadmium; FAD NAD P H; GSSG GSH; HepG2 cells; Oxidative stress; ROS

Indexed keywords

CADMIUM CHLORIDE; FLAVINE ADENINE NUCLEOTIDE; GLUTATHIONE; GLUTATHIONE DISULFIDE; PROTEIN; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE;

ARTICLE; AUTOFLUORESCENCE; BINDING SITE; CELL STRAIN HEPG2; CELL STRESS; COMPARATIVE STUDY; CONFOCAL MICROSCOPY; CONTROLLED STUDY; DRUG EXPOSURE; ELECTRON TRANSPORT; ENZYME ACTIVITY; HUMAN; HUMAN CELL; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN BINDING; TIME;

BINDING SITES; CADMIUM CHLORIDE; CELL LINE, TUMOR; FLAVIN-ADENINE DINUCLEOTIDE; GLUTATHIONE; GLUTATHIONE DISULFIDE; HUMANS; NADP; OXIDATIVE STRESS; PROTEIN BINDING; REACTIVE OXYGEN SPECIES; SPECTROMETRY, FLUORESCENCE; TIME FACTORS;

EID: 41449113683     PISSN: 0300483X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.tox.2008.01.021     Document Type: Article

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