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Volumn 586, Issue 4, 2008, Pages 923-
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Evidence of a physiological role for use-dependent inactivation of NaV1.8 sodium channels
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Author keywords
[No Author keywords available]
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Indexed keywords
ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM);
ANALGESIC AGENT;
ANTICONVULSIVE AGENT;
CARBAMAZEPINE;
LIDOCAINE;
SODIUM CHANNEL;
SODIUM CHANNEL NAV1.8;
TETRODOTOXIN;
UNCLASSIFIED DRUG;
PROSTAGLANDIN E2;
SEROTONIN;
ARTICLE;
ENZYME INHIBITION;
NERVE STIMULATION;
NEUROTRANSMISSION;
NOCICEPTIVE RECEPTOR;
NONHUMAN;
NONMYELINATED NERVE;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
PROTEIN FUNCTION;
SENSORY NERVE CONDUCTION;
SPINAL CORD NERVE CELL;
DRUG TARGETING;
FIRING RATE;
KINETIC PARAMETERS;
NERVE CONDUCTION;
NERVE POTENTIAL;
NOTE;
PAIN;
PAIN RECEPTOR;
SENSORY NERVE CELL;
SPINAL GANGLION;
USE DEPENDENT INACTIVATION;
ACTION POTENTIALS;
ANIMALS;
NERVE TISSUE PROTEINS;
NOCICEPTORS;
RATS;
SODIUM CHANNELS;
SYNAPTIC TRANSMISSION;
TETRODOTOXIN;
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EID: 39449118864
PISSN: 00223751
EISSN: 14697793
Source Type: Journal
DOI: 10.1113/jphysiol.2008.150821 Document Type: Article |
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Times cited : (3)
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References (5)
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