-
1
-
-
18044391352
-
The neutralization of FGF-23 ameliorates hypophosphatemia and rickets in Hyp mice (Abstract)
-
Aono Y, Shimada T, Uamazaki Y, Hino R, Takeuchi Y, Fujita T, Fukumoto S, Nagano N, Wada M, Yamashita T. The neutralization of FGF-23 ameliorates hypophosphatemia and rickets in Hyp mice (Abstract). J Bone Miner Res 18: S16, 2003.
-
(2003)
J Bone Miner Res
, vol.18
-
-
Aono, Y.1
Shimada, T.2
Uamazaki, Y.3
Hino, R.4
Takeuchi, Y.5
Fujita, T.6
Fukumoto, S.7
Nagano, N.8
Wada, M.9
Yamashita, T.10
-
2
-
-
9244240970
-
Transgenic mice overexpressing human fibroblast growth factor 23 (R176Q) delineate a putative role for parathyroid hormone in renal phosphate wasting disorders
-
Bai X, Miao D, Li J, Goltzman D, Karaplis AC. Transgenic mice overexpressing human fibroblast growth factor 23 (R176Q) delineate a putative role for parathyroid hormone in renal phosphate wasting disorders. Endocrinology 145: 5269-5279, 2004.
-
(2004)
Endocrinology
, vol.145
, pp. 5269-5279
-
-
Bai, X.1
Miao, D.2
Li, J.3
Goltzman, D.4
Karaplis, A.C.5
-
3
-
-
0013118819
-
The autosomal dominant hypophosphatemic rickets R176Q mutation in fibroblast growth factor 23 resists proteolytic cleavage and enhances in vivo biological potency
-
Bai XY, Miao D, Goltzman D, Karaplis AC. The autosomal dominant hypophosphatemic rickets R176Q mutation in fibroblast growth factor 23 resists proteolytic cleavage and enhances in vivo biological potency. J Biol Chem 278: 9843-9849, 2003.
-
(2003)
J Biol Chem
, vol.278
, pp. 9843-9849
-
-
Bai, X.Y.1
Miao, D.2
Goltzman, D.3
Karaplis, A.C.4
-
4
-
-
0030938927
-
Pex/PEX tissue distribution and evidence for a deletion in the 3′ region of the Pex gene in X-linked hypophosphatemic mice
-
Beck L, Soumounou Y, Martel J, Krishnamurthy G, Gauthier C, Goodyer CG, Tenenhouse HS. Pex/PEX tissue distribution and evidence for a deletion in the 3′ region of the Pex gene in X-linked hypophosphatemic mice. J Clin Invest 99: 1200-1209, 1997.
-
(1997)
J Clin Invest
, vol.99
, pp. 1200-1209
-
-
Beck, L.1
Soumounou, Y.2
Martel, J.3
Krishnamurthy, G.4
Gauthier, C.5
Goodyer, C.G.6
Tenenhouse, H.S.7
-
5
-
-
3242656464
-
FGF23 is processed by proprotein convertases but not by PHEX
-
Benet-Pages A, Lorenz-Depiereux B, Zischka H, White KE, Econs MJ, Strom TM. FGF23 is processed by proprotein convertases but not by PHEX. Bone 35: 455-462, 2004.
-
(2004)
Bone
, vol.35
, pp. 455-462
-
-
Benet-Pages, A.1
Lorenz-Depiereux, B.2
Zischka, H.3
White, K.E.4
Econs, M.J.5
Strom, T.M.6
-
6
-
-
0034805353
-
FGF-23 inhibits renal tubular phosphate transport and is a PHEX substrate
-
Bowe AE, Finnegan R, Jan de Beur SM, Cho J, Levine MA, Kumar R, Schiavi SC. FGF-23 inhibits renal tubular phosphate transport and is a PHEX substrate. Biochem Biophys Res Commun 284: 977-981, 2001.
-
(2001)
Biochem Biophys Res Commun
, vol.284
, pp. 977-981
-
-
Bowe, A.E.1
Finnegan, R.2
Jan de Beur, S.M.3
Cho, J.4
Levine, M.A.5
Kumar, R.6
Schiavi, S.C.7
-
7
-
-
0026513193
-
Defective bone formation by Hyp mouse bone cells transplanted into normal mice: Evidence in favor of an intrinsic osteoblast defect
-
Ecarot B, Glorieux FH, Desbarats M, Travers R, Labelle L. Defective bone formation by Hyp mouse bone cells transplanted into normal mice: evidence in favor of an intrinsic osteoblast defect. J Bone Miner Res 7: 215-220, 1992.
-
(1992)
J Bone Miner Res
, vol.7
, pp. 215-220
-
-
Ecarot, B.1
Glorieux, F.H.2
Desbarats, M.3
Travers, R.4
Labelle, L.5
-
8
-
-
33750454816
-
Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism
-
Feng JQ, Ward LM, Liu S, Lu Y, Xie Y, Yuan B, Yu X, Rauch F, Davis SI, Zhang S, Rios H, Drezner MK, Quarles LD, Bonewald LF, White KE. Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism. Nat Genet 38: 1310-1315, 2006.
-
(2006)
Nat Genet
, vol.38
, pp. 1310-1315
-
-
Feng, J.Q.1
Ward, L.M.2
Liu, S.3
Lu, Y.4
Xie, Y.5
Yuan, B.6
Yu, X.7
Rauch, F.8
Davis, S.I.9
Zhang, S.10
Rios, H.11
Drezner, M.K.12
Quarles, L.D.13
Bonewald, L.F.14
White, K.E.15
-
9
-
-
34249664523
-
FGF23 concentrations vary with disease status in autosomal dominant hypophosphatemic rickets
-
Imel EA, Hui SL, Econs MJ. FGF23 concentrations vary with disease status in autosomal dominant hypophosphatemic rickets. J Bone Miner Res 22: 520-526, 2007.
-
(2007)
J Bone Miner Res
, vol.22
, pp. 520-526
-
-
Imel, E.A.1
Hui, S.L.2
Econs, M.J.3
-
10
-
-
33646578195
-
Regulation of fibroblast growth factor-23 signaling by klotho
-
Kurosu H, Ogawa Y, Miyoshi M, Yamamoto M, Nandi A, Rosenblatt KP, Baum MG, Schiavi S, Hu MC, Moe OW, Kuro-o M. Regulation of fibroblast growth factor-23 signaling by klotho. J Biol Chem 281: 6120-6123, 2006.
-
(2006)
J Biol Chem
, vol.281
, pp. 6120-6123
-
-
Kurosu, H.1
Ogawa, Y.2
Miyoshi, M.3
Yamamoto, M.4
Nandi, A.5
Rosenblatt, K.P.6
Baum, M.G.7
Schiavi, S.8
Hu, M.C.9
Moe, O.W.10
Kuro-o, M.11
-
11
-
-
23844457598
-
Fibroblast growth factor-23 mutants causing familial tumoral calcinosis are differentially processed
-
Larsson T, Davis SI, Garringer HJ, Mooney SD, Draman MS, Cullen MJ, White KE. Fibroblast growth factor-23 mutants causing familial tumoral calcinosis are differentially processed. Endocrinology 146: 3883-3891, 2005.
-
(2005)
Endocrinology
, vol.146
, pp. 3883-3891
-
-
Larsson, T.1
Davis, S.I.2
Garringer, H.J.3
Mooney, S.D.4
Draman, M.S.5
Cullen, M.J.6
White, K.E.7
-
12
-
-
3042634460
-
Transgenic mice expressing fibroblast growth factor 23 under the control of the α1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasis
-
Larsson T, Marsell R, Schipani E, Ohlsson C, Ljunggren O, Tenenhouse HS, Juppner H, Jonsson KB. Transgenic mice expressing fibroblast growth factor 23 under the control of the α1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasis. Endocrinology 145: 3087-3094, 2004.
-
(2004)
Endocrinology
, vol.145
, pp. 3087-3094
-
-
Larsson, T.1
Marsell, R.2
Schipani, E.3
Ohlsson, C.4
Ljunggren, O.5
Tenenhouse, H.S.6
Juppner, H.7
Jonsson, K.B.8
-
13
-
-
0141844575
-
Regulation of fibroblastic growth factor 23 expression but not degradation by PHEX
-
Liu S, Guo R, Simpson LG, Xiao ZS, Burnham CE, Quarles LD. Regulation of fibroblastic growth factor 23 expression but not degradation by PHEX. J Biol Chem 278: 37419-37426, 2003.
-
(2003)
J Biol Chem
, vol.278
, pp. 37419-37426
-
-
Liu, S.1
Guo, R.2
Simpson, L.G.3
Xiao, Z.S.4
Burnham, C.E.5
Quarles, L.D.6
-
14
-
-
34249883939
-
How fibroblast growth factor 23 works
-
Liu S, Quarles LD. How fibroblast growth factor 23 works. J Am Soc Nephrol 18: 1637-1647, 2007.
-
(2007)
J Am Soc Nephrol
, vol.18
, pp. 1637-1647
-
-
Liu, S.1
Quarles, L.D.2
-
15
-
-
33745850756
-
Pathogenic role of Fgf23 in Hyp mice
-
Liu S, Zhou J, Tang W, Jiang X, Rowe DW, Quarles LD. Pathogenic role of Fgf23 in Hyp mice. Am J Physiol Endocrinol Metab 291: E38-E49, 2006.
-
(2006)
Am J Physiol Endocrinol Metab
, vol.291
-
-
Liu, S.1
Zhou, J.2
Tang, W.3
Jiang, X.4
Rowe, D.W.5
Quarles, L.D.6
-
16
-
-
33750427897
-
DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis
-
Lorenz-Depiereux B, Bastepe M, Benet-Pages A, Amyere M, Wagenstaller J, Muller-Barth U, Badenhoop K, Kaiser SM, Rittmaster RS, Shlossberg AH, Olivares JL, Loris C, Ramos FJ, Glorieux F, Vikkula M, Juppner H, Strom TM. DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis. Nat Genet 38: 1248-1250, 2006.
-
(2006)
Nat Genet
, vol.38
, pp. 1248-1250
-
-
Lorenz-Depiereux, B.1
Bastepe, M.2
Benet-Pages, A.3
Amyere, M.4
Wagenstaller, J.5
Muller-Barth, U.6
Badenhoop, K.7
Kaiser, S.M.8
Rittmaster, R.S.9
Shlossberg, A.H.10
Olivares, J.L.11
Loris, C.12
Ramos, F.J.13
Glorieux, F.14
Vikkula, M.15
Juppner, H.16
Strom, T.M.17
-
17
-
-
33845296928
-
Vitamin D receptor in chondrocytes promotes osteoclastogenesis and regulates FGF23 production in osteoblasts
-
Masuyama R, Stockmans I, Torrekens S, Van Looveren R, Maes C, Carmeliet P, Bouillon R, Carmeliet G. Vitamin D receptor in chondrocytes promotes osteoclastogenesis and regulates FGF23 production in osteoblasts. J Clin Invest 116: 3150-3159, 2006.
-
(2006)
J Clin Invest
, vol.116
, pp. 3150-3159
-
-
Masuyama, R.1
Stockmans, I.2
Torrekens, S.3
Van Looveren, R.4
Maes, C.5
Carmeliet, P.6
Bouillon, R.7
Carmeliet, G.8
-
18
-
-
0035107364
-
Osteomalacia in hyp mice is associated with abnormal phex expression and with altered bone matrix protein expression and deposition
-
Miao D, Bai X, Panda D, McKee M, Karaplis A, Goltzman D. Osteomalacia in hyp mice is associated with abnormal phex expression and with altered bone matrix protein expression and deposition. Endocrinology 142: 926-939, 2001.
-
(2001)
Endocrinology
, vol.142
, pp. 926-939
-
-
Miao, D.1
Bai, X.2
Panda, D.3
McKee, M.4
Karaplis, A.5
Goltzman, D.6
-
19
-
-
1642305862
-
Cartilage abnormalities are associated with abnormal Phex expression and with altered matrix protein and MMP-9 localization in Hyp mice
-
Miao D, Bai X, Panda DK, Karaplis AC, Goltzman D, McKee MD. Cartilage abnormalities are associated with abnormal Phex expression and with altered matrix protein and MMP-9 localization in Hyp mice. Bone 34: 638-647, 2004.
-
(2004)
Bone
, vol.34
, pp. 638-647
-
-
Miao, D.1
Bai, X.2
Panda, D.K.3
Karaplis, A.C.4
Goltzman, D.5
McKee, M.D.6
-
20
-
-
18244392685
-
Unique coexpression in osteoblasts of broadly expressed genes accounts for the spatial restriction of ECM mineralization to bone
-
Murshed M, Harmey D, Millan JL, McKee MD, Karsenty G. Unique coexpression in osteoblasts of broadly expressed genes accounts for the spatial restriction of ECM mineralization to bone. Genes Dev 19: 1093-1104, 2005.
-
(2005)
Genes Dev
, vol.19
, pp. 1093-1104
-
-
Murshed, M.1
Harmey, D.2
Millan, J.L.3
McKee, M.D.4
Karsenty, G.5
-
21
-
-
28744444868
-
Dentinal defects in Hyp mice not caused by hypophosphatemia alone
-
Ogawa T, Onishi T, Hayashibara T, Sakashita S, Okawa R, Ooshima T. Dentinal defects in Hyp mice not caused by hypophosphatemia alone. Arch Oral Biol 51: 58-63, 2006.
-
(2006)
Arch Oral Biol
, vol.51
, pp. 58-63
-
-
Ogawa, T.1
Onishi, T.2
Hayashibara, T.3
Sakashita, S.4
Okawa, R.5
Ooshima, T.6
-
22
-
-
0026752938
-
Effects of etidronate-mediated suppression of bone remodeling on aluminum-induced de novo bone formation
-
Quarles LD, Drezner MK. Effects of etidronate-mediated suppression of bone remodeling on aluminum-induced de novo bone formation. Endocrinology 131: 122-126, 1992.
-
(1992)
Endocrinology
, vol.131
, pp. 122-126
-
-
Quarles, L.D.1
Drezner, M.K.2
-
23
-
-
0025974476
-
X-linked hypophosphatemia: Skeletal mass in adults assessed by histomorphometry, computed tomography, and absorptiometry
-
Reid IR, Murphy WA, Hardy DC, Teitelbaum SL, Bergfeld MA, Whyte MP. X-linked hypophosphatemia: skeletal mass in adults assessed by histomorphometry, computed tomography, and absorptiometry. Am J Med 90: 63-69, 1991.
-
(1991)
Am J Med
, vol.90
, pp. 63-69
-
-
Reid, I.R.1
Murphy, W.A.2
Hardy, D.C.3
Teitelbaum, S.L.4
Bergfeld, M.A.5
Whyte, M.P.6
-
24
-
-
2142746439
-
FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis
-
Shimada T, Hasegawa H, Yamazaki Y, Muto T, Hino R, Takeuchi Y, Fujita T, Nakahara K, Fukumoto S, Yamashita T. FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis. J Bone Miner Res 19: 429-435, 2004.
-
(2004)
J Bone Miner Res
, vol.19
, pp. 429-435
-
-
Shimada, T.1
Hasegawa, H.2
Yamazaki, Y.3
Muto, T.4
Hino, R.5
Takeuchi, Y.6
Fujita, T.7
Nakahara, K.8
Fukumoto, S.9
Yamashita, T.10
-
25
-
-
1642416884
-
Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism
-
Shimada T, Kakitani M, Yamazaki Y, Hasegawa H, Takeuchi Y, Fujita T, Fukumoto S, Tomizuka K, Yamashita T. Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism. J Clin Invest 113: 561-568, 2004.
-
(2004)
J Clin Invest
, vol.113
, pp. 561-568
-
-
Shimada, T.1
Kakitani, M.2
Yamazaki, Y.3
Hasegawa, H.4
Takeuchi, Y.5
Fujita, T.6
Fukumoto, S.7
Tomizuka, K.8
Yamashita, T.9
-
26
-
-
14344279878
-
Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia
-
Shimada T, Mizutani S, Muto T, Yoneya T, Hino R, Takeda S, Takeuchi Y, Fujita T, Fukumoto S, Yamashita T. Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia. Proc Natl Acad Sci USA 98: 6500-6505, 2001.
-
(2001)
Proc Natl Acad Sci USA
, vol.98
, pp. 6500-6505
-
-
Shimada, T.1
Mizutani, S.2
Muto, T.3
Yoneya, T.4
Hino, R.5
Takeda, S.6
Takeuchi, Y.7
Fujita, T.8
Fukumoto, S.9
Yamashita, T.10
-
27
-
-
18444375871
-
Mutant FGF-23 responsible for autosomal dominant hypophosphatemic rickets is resistant to proteolytic cleavage and causes hypophosphatemia in vivo
-
Shimada T, Muto T, Urakawa I, Yoneya T, Yamazaki Y, Okawa K, Takeuchi Y, Fujita T, Fukumoto S, Yamashita T. Mutant FGF-23 responsible for autosomal dominant hypophosphatemic rickets is resistant to proteolytic cleavage and causes hypophosphatemia in vivo. Endocrinology 143: 3179-3182, 2002.
-
(2002)
Endocrinology
, vol.143
, pp. 3179-3182
-
-
Shimada, T.1
Muto, T.2
Urakawa, I.3
Yoneya, T.4
Yamazaki, Y.5
Okawa, K.6
Takeuchi, Y.7
Fujita, T.8
Fukumoto, S.9
Yamashita, T.10
-
28
-
-
9644303231
-
Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice
-
Sitara D, Razzaque MS, Hesse M, Yoganathan S, Taguchi T, Erben RG, Juppner H, Lanske B. Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice. Matrix Biol 23: 421-432, 2004.
-
(2004)
Matrix Biol
, vol.23
, pp. 421-432
-
-
Sitara, D.1
Razzaque, M.S.2
Hesse, M.3
Yoganathan, S.4
Taguchi, T.5
Erben, R.G.6
Juppner, H.7
Lanske, B.8
-
29
-
-
0030615069
-
Pex gene deletions in Gy and Hyp mice provide mouse models for X-linked hypophosphatemia
-
Strom TM, Francis F, Lorenz B, Boddrich A, Econs MJ, Lehrach H, Meitinger T. Pex gene deletions in Gy and Hyp mice provide mouse models for X-linked hypophosphatemia. Hum Mol Genet 6: 165-171, 1997.
-
(1997)
Hum Mol Genet
, vol.6
, pp. 165-171
-
-
Strom, T.M.1
Francis, F.2
Lorenz, B.3
Boddrich, A.4
Econs, M.J.5
Lehrach, H.6
Meitinger, T.7
-
30
-
-
3042590141
-
Direct action of 1,25-dihydroxyvitamin D on bone: VDRKO bone shows excessive bone formation in normal mineral condition
-
Tanaka H, Seino Y. Direct action of 1,25-dihydroxyvitamin D on bone: VDRKO bone shows excessive bone formation in normal mineral condition. J Steroid Biochem Mol Biol 89-90: 343-345, 2004.
-
(2004)
J Steroid Biochem Mol Biol
, vol.89-90
, pp. 343-345
-
-
Tanaka, H.1
Seino, Y.2
-
31
-
-
0033763097
-
Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23
-
The ADHR Consortium
-
The ADHR Consortium. Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23. Nat Genet 26: 345-348, 2000.
-
(2000)
Nat Genet
, vol.26
, pp. 345-348
-
-
-
32
-
-
0029160578
-
A gene (PEX) with homologies to endopeptidases is mutated in patients with X-linked hypophosphatemic rickets
-
The HYP Consortium
-
The HYP Consortium. A gene (PEX) with homologies to endopeptidases is mutated in patients with X-linked hypophosphatemic rickets. Nat Genet 11: 130-136, 1995.
-
(1995)
Nat Genet
, vol.11
, pp. 130-136
-
-
-
33
-
-
0030899822
-
Mammalian membrane metallopeptidases: NEP, ECE, KELL, and PEX
-
Turner AJ, Tanzawa K. Mammalian membrane metallopeptidases: NEP, ECE, KELL, and PEX. FASEB J 11: 355-364, 1997.
-
(1997)
FASEB J
, vol.11
, pp. 355-364
-
-
Turner, A.J.1
Tanzawa, K.2
-
34
-
-
33845631059
-
Klotho converts canonical FGF receptor into a specific receptor for FGF23
-
Urakawa I, Yamazaki Y, Shimada T, Iijima K, Hasegawa H, Okawa K, Fujita T, Fukumoto S, Yamashita T. Klotho converts canonical FGF receptor into a specific receptor for FGF23. Nature 444: 770-774, 2006.
-
(2006)
Nature
, vol.444
, pp. 770-774
-
-
Urakawa, I.1
Yamazaki, Y.2
Shimada, T.3
Iijima, K.4
Hasegawa, H.5
Okawa, K.6
Fujita, T.7
Fukumoto, S.8
Yamashita, T.9
-
35
-
-
0141525564
-
Serum FGF23 levels in normal and disordered phosphorus homeostasis
-
Weber TJ, Liu S, Indridason OS, Quarles LD. Serum FGF23 levels in normal and disordered phosphorus homeostasis. J Bone Miner Res 18: 1227-1234, 2003.
-
(2003)
J Bone Miner Res
, vol.18
, pp. 1227-1234
-
-
Weber, T.J.1
Liu, S.2
Indridason, O.S.3
Quarles, L.D.4
-
36
-
-
0035186837
-
Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23
-
White KE, Carn G, Lorenz-Depiereux B, Benet-Pages A, Strom TM, Econs MJ. Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23. Kidney Int 60: 2079-2086, 2001.
-
(2001)
Kidney Int
, vol.60
, pp. 2079-2086
-
-
White, K.E.1
Carn, G.2
Lorenz-Depiereux, B.3
Benet-Pages, A.4
Strom, T.M.5
Econs, M.J.6
-
37
-
-
17744395066
-
The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting
-
White KE, Jonsson KB, Carn G, Hampson G, Spector TD, Mannstadt M, Lorenz-Depiereux B, Miyauchi A, Yang IM, Ljunggren O, Meitinger T, Strom TM, Juppner H, Econs MJ. The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting. J Clin Endocrinol Metab 86: 497-500, 2001.
-
(2001)
J Clin Endocrinol Metab
, vol.86
, pp. 497-500
-
-
White, K.E.1
Jonsson, K.B.2
Carn, G.3
Hampson, G.4
Spector, T.D.5
Mannstadt, M.6
Lorenz-Depiereux, B.7
Miyauchi, A.8
Yang, I.M.9
Ljunggren, O.10
Meitinger, T.11
Strom, T.M.12
Juppner, H.13
Econs, M.J.14
-
38
-
-
0031796266
-
Intrinsic mineralization defect in Hyp mouse osteoblasts
-
Xiao ZS, Crenshaw M, Guo R, Nesbitt T, Drezner MK, Quarles LD. Intrinsic mineralization defect in Hyp mouse osteoblasts. Am J Physiol Endocrinol Metab 275: E700-E708, 1998.
-
(1998)
Am J Physiol Endocrinol Metab
, vol.275
-
-
Xiao, Z.S.1
Crenshaw, M.2
Guo, R.3
Nesbitt, T.4
Drezner, M.K.5
Quarles, L.D.6
-
39
-
-
18744371012
-
Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia
-
Yamazaki Y, Okazaki R, Shibata M, Hasegawa Y, Satoh K, Tajima T, Takeuchi Y, Fujita T, Nakahara K, Yamashita T, Fukumoto S. Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia. J Clin Endocrinol Metab 87: 4957-4960, 2002.
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(2002)
J Clin Endocrinol Metab
, vol.87
, pp. 4957-4960
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Yamazaki, Y.1
Okazaki, R.2
Shibata, M.3
Hasegawa, Y.4
Satoh, K.5
Tajima, T.6
Takeuchi, Y.7
Fujita, T.8
Nakahara, K.9
Yamashita, T.10
Fukumoto, S.11
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