Senescence regulates macrophage activation and angiogenic fate at sites of tissue injury in mice

Journal of Clinical Investigation

Volumn 117, Issue 11, 2007, Pages 3421-3426

  Kelly, Jennifer ^a   Khan, Aslam Ali ^a   Yin, Jiyi ^a   Ferguson, Thomas A ^a   Apte, Rajendra S ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; FAS LIGAND; INTERLEUKIN 10; INTERLEUKIN 12; TUMOR NECROSIS FACTOR ALPHA;

ANGIOGENESIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLINDNESS; BLOOD VESSEL; CANCER; CELL PROLIFERATION; CONTROLLED STUDY; DISEASE COURSE; ENDOTHELIUM CELL; EYE DISEASE; GENE EXPRESSION; HEART DISEASE; LASER; MACROPHAGE; MACROPHAGE ACTIVATION; MACROPHAGE FUNCTION; MOUSE; NONHUMAN; PHENOTYPE; POLARIZATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; REAL TIME POLYMERASE CHAIN REACTION; RETINA INJURY; RETINA MACULA AGE RELATED DEGENERATION; RISK FACTOR; SENESCENCE; TISSUE INJURY; VASCULAR ENDOTHELIUM;

AGING; ANIMALS; CELL AGING; CHOROIDAL NEOVASCULARIZATION; CYTOKINES; GENE EXPRESSION REGULATION; HUMANS; INTERLEUKIN-10; LASERS; MACROPHAGE ACTIVATION; MACROPHAGES; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; RETINA;

EID: 36048953291     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI32430     Document Type: Article

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