One potential mechanism of cardiac toxicity induced by arsenic trioxide: Apoptosis in coronary arterial and blood capillary endothelial cells

Medical Hypotheses

Volumn 69, Issue 6, 2007, Pages 1383-1384

  Liu, Yu ^a   Cai, Benzhi ^a   Wang, Guonian ^a   Liu, Jinfeng ^a  

^a HARBIN MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ARSENIC TRIOXIDE;

APOPTOSIS; CAPILLARY ENDOTHELIUM; CARDIOTOXICITY; CELL DAMAGE; CELL PROLIFERATION; CELL PROTECTION; CORONARY ARTERY; DRUG EFFECT; DRUG MECHANISM; ENDOTHELIUM CELL; HUMAN; HYPOTHESIS; LETTER; PRIORITY JOURNAL; PROMYELOCYTIC LEUKEMIA;

ANIMALS; APOPTOSIS; ARSENICALS; CAPILLARIES; CATTLE; ENDOTHELIAL CELLS; HEART; HUMANS; MODELS, BIOLOGICAL; MODELS, THEORETICAL; MYOCARDIUM; OXIDES; SIGNAL TRANSDUCTION; UMBILICAL VEINS;

EID: 35448993992     PISSN: 03069877     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mehy.2007.03.009     Document Type: Letter

References (7)

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