Burn injury decreases myocardial Na-K-ATPase activity: Role of PKC inhibition

American Journal of Physiology - Regulatory Integrative and Comparative Physiology

Volumn 293, Issue 4, 2007, Pages

  Horton, Jureta W ^a   Tan, Jing ^a   White, D Jean ^a   Maass, David L ^a  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

Calphostin; Myocyte sodium; Protein kinase C activity; Protein kinase C inhibitor; Rat burn injury model; Ventricular function

Indexed keywords

ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM); ADENOSINE TRIPHOSPHATE; CALPHOSTIN; OUABAIN; PHOSPHATE; PROTEIN KINASE C; RINGER LACTATE SOLUTION; SODIUM ION;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BODY SURFACE; BURN; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME INHIBITION; HEART LEFT VENTRICLE PERFORMANCE; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HISTOCHEMISTRY; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; SARCOLEMMA;

ANIMALS; BURNS; CYTOKINES; ENZYME INHIBITORS; MALE; MYOCARDIAL CONTRACTION; MYOCARDIUM; MYOCYTES, CARDIAC; NAPHTHALENES; PROTEIN KINASE C; RATS; RATS, SPRAGUE-DAWLEY; SODIUM; SODIUM-POTASSIUM-EXCHANGING ATPASE;

EID: 35148849230     PISSN: 03636119     EISSN: 15221490     Source Type: Journal    
DOI: 10.1152/ajpregu.00219.2007     Document Type: Article

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