Isoniazid induces oxidative stress, mitochondrial dysfunction and apoptosis in Hep G2 cells

Cellular and Molecular Biology

Volumn 53, Issue 1, 2007, Pages 102-114

  Bhadauria, S ^a   Singh, G ^b   Sinha, N ^b   Srivastava, S ^b  

^a CENTRAL DRUG RESEARCH INSTITUTE   (India)

^b NONE

Author keywords

Apoptosis; Hepatotoxicity; Oxidative stress; Reactive oxygen species

Indexed keywords

GLUTATHIONE; ISONIAZID; REACTIVE OXYGEN METABOLITE;

APOPTOSIS; ARTICLE; CELL SIZE; CELL STRUCTURE; CELL VIABILITY; CONTROLLED STUDY; CYTOTOXICITY; DNA DETERMINATION; DOSE RESPONSE; EXPOSURE; FLOW CYTOMETRY; HUMAN; HUMAN CELL; MITOCHONDRION; OXIDATIVE STRESS;

ANIMALS; ANTITUBERCULAR AGENTS; APOPTOSIS; CARCINOMA, HEPATOCELLULAR; CELL LINE, TUMOR; CELL SHAPE; CELL SURVIVAL; CYTOPLASM; DNA FRAGMENTATION; DOSE-RESPONSE RELATIONSHIP, DRUG; GLUTATHIONE; HUMANS; ISONIAZID; L-LACTATE DEHYDROGENASE; LIVER NEOPLASMS; MITOCHONDRIA; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES;

EID: 34249083462     PISSN: 01455680     EISSN: 1165158X     Source Type: Journal    
DOI: 10.1170/T781     Document Type: Article

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