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Volumn 64, Issue 5, 2007, Pages 643-646

Genetic models of migraine

Author keywords

[No Author keywords available]

Indexed keywords

BRAIN DISEASE; BRAIN FUNCTION; CALCIUM TRANSPORT; CLINICAL FEATURE; EXPERIMENTAL MODEL; FHM2 GENE; FHM3 GENE; GENE; GENE MUTATION; GENETIC MODEL; GENETIC SUSCEPTIBILITY; HEADACHE; HEART FUNCTION TEST; HEMIPLEGIA; HEREDITY; MIGRAINE; MIGRAINE AURA; NONHUMAN; PRIORITY JOURNAL; REVIEW;

EID: 34249002104     PISSN: 00039942     EISSN: 15383687     Source Type: Journal    
DOI: 10.1001/archneur.64.5.643     Document Type: Review
Times cited : (56)

References (13)
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  • 3
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  • 4
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    • Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4
    • Ophoff RA, Terwindt GM, Vergouwe MN, et al. Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4. Cell. 1996;87:543-552.
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  • 5
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    • De Fusco, M.1    Marconi, R.2    Silvestri, L.3
  • 6
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    • Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine
    • Dichgans M, Freilinger T, Eckstein G, et al. Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine. Lancet. 2005;366:371-377.
    • (2005) Lancet , vol.366 , pp. 371-377
    • Dichgans, M.1    Freilinger, T.2    Eckstein, G.3
  • 8
    • 0842282679 scopus 로고    scopus 로고
    • Deciphering migraine mechanisms: Clues from familial hemiplegic migraine genotypes
    • Moskowitz MA, Bolay H, Dalkara T. Deciphering migraine mechanisms: clues from familial hemiplegic migraine genotypes. Ann Neurol. 2004;55:276-280.
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  • 9
    • 12144286750 scopus 로고    scopus 로고
    • A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression
    • van den Maagdenberg AM, Pietrobon D, Pizzorusso T, et al. A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression. Neuron. 2004;41:701-710.
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  • 10
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    • Gene dosage-dependent transmitter release changes at neuromuscular synapses of Cacna1a R192Q knockin mice are non-progressive and do not lead to morphological changes or muscle weakness
    • Kaja S, van de Ven RC, Broos LA, et al. Gene dosage-dependent transmitter release changes at neuromuscular synapses of Cacna1a R192Q knockin mice are non-progressive and do not lead to morphological changes or muscle weakness. Neuroscience. 2005;135:81-95.
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    • Delayed cerebral edema and fatal coma after minor head trauma: Role of the CACNA1A calcium channel subunit gene and relationship with familial hemiplegic migraine
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  • 12
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    • Migraine as a risk factor for subclinical brain lesions
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  • 13
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.